What is ketoacidosis in the first trimester? How can I avoid it?

It is possible that the pregnant woman has gestational diabetes during pregnancy, or it is possible that the pregnancy has a severe reaction and electrolyte imbalance leading to poisoning. Drink plenty of water, eat plenty of vegetables and fruits, and get plenty of rest in the early stages of pregnancy.

May be caused by severe vomiting. It is an acute metabolic disease caused by severe insulin deficiency in pregnant women. Drink plenty of water and check your blood sugar in time to avoid this phenomenon.

It can be due to severe vomiting. It is an acute metabolic disease caused by a severe shortage of insulin in the pregnant woman's body. Drink plenty of water and check your blood sugar in time to avoid this phenomenon.

Ketoacidosis during pregnancy is an acute metabolic complication caused by a severe shortage of insulin in the pregnant woman's body, and as a high-risk group for diabetes, this condition can be life-threatening for the pregnant woman and the fetus if not prompted**. If you have diabetes during pregnancy, you must go to the hospital for regular check-ups. Moreover, after the diagnosis, it is necessary to adjust the diet and exercise under the guidance of the doctor to do the corresponding **.

Generally, the acid substances in the ketone body accumulate too much in the blood, and the blood becomes acidic for acidosis, so supplement vitamins and drink more water to detoxify.

Ketoacidosis refers to metabolic acidosis associated with ketone accumulation. Basic** can be divided into diabetic ketoacidosis and alcoholic ketoacidosis and starvation ketoacidosis. Don't over-diet and maintain good habits.

The common triggers are as follows: 1. Insufficient insulin dosage and unreasonable regimen; 2. Infection: such as lung infection or infection in other parts; 3. Poor diet control; 4. Stress factors:

Such as surgery, etc. Six precautions: 1. Absolute bed rest after being diagnosed with ketoacidosis, and rescue should be cooperated immediately**.

2. Quickly establish intravenous access to correct water, electrolyte and acid-base balance disorders and ketoacidosis symptoms. 3. Follow the doctor's instructions and use regular insulin. Low-dose insulin should be used correctly to reduce the occurrence of hypoglycemia, hypokalemia and cerebral edema4, assist in inducing diseases and complications, closely observe the patient's vital signs, consciousness, and pupils (see Coma Care Procedure), and assist in the measurement and recording of blood glucose.

5. Diet care fasting, and change to diabetic semi-liquid or diabetic diet after coma is relieved. 6. To prevent infection, we must do a good job of oral and ** care and keep ** clean.

Healthy and light: fruit meal replacement is easy to kill? Ketoacidosis, what's going on?

Rehydration. The first and most critical measure to rescue a patient with ketoacidosis is to give adequate fluids to help restore blood volume, increase tissue perfusion, and facilitate insulin transport to peripheral tissues. By increasing renal blood perfusion, the excretion of glucose in the urine is increased, which can reduce blood sugar.

Principle of rehydration: when the blood glucose is greater than 167 milliliters, normal saline should be used in time, and intravenous infusion should be given at a rate of 500-1000 ml per hour; When the blood glucose is milliliters, it can be switched to intravenous infusion of glucose solution, and the speed is slowed down. The dose of fluids can be given at the rate of 10% of body weight.

Insulin injections.

Insulin is the key drug for ketoacidosis. Common medication regimens:

a. Those with high blood sugar.

mmol L), insulin first.

Once-in-one intravenous injection.

b. Continuous intravenous infusion of insulin

Sodium chloride injection.

Insulin, press insulin.

u/(kg•h)

or 4-6u h

speed input.

c. When the blood sugar drops.

mmol l, will.

Sodium chloride injection was changed to.

Dextrose solution or dextrose saline, each.

Add grams of glucose.

1 u of insulin until blood sugar drops.

mmol/l

Below, urine ketones are negative, and fluid replacement can be stopped when there is a smooth transition to preprandial subcutaneous injection**.

Potassium supplementation. Serum potassium is always low in ketoacidosis, so potassium deficiency should be noted at the same time as fluid renewal, and potassium replacement should be given at the outset, with close supervision of potassium and ECG.

In general, blood potassium is in.

mmol/l

may not be given potassium supplementation for the time being, but serum potassium should be closely monitored once it is lower.

mmol/l

Potassium supplementation is required**.

Alkali supplementation. Patients with ketoacidosis have a pH of carbon dioxide binding <10

mmol/l、hco3-<10

In the case of mmol L, alkali supplementation is also required**.

5% NaHCO3- is generally used

ml + water for injection 400

ml, to 200

Intravenous infusion at the rate of ml H, to pH

or CO2 binding" 15

mmol/l

Stop alkali supplementation.