Is otolithiasis related to tinnitus? What is otolithiasis?

Tinnitus is a complication of many diseases, otolithiasis itself cannot cause tinnitus, and one of the pathological structures of otolithiasis is the oval sac and the semicircular canal, which are different structures. Some people are accompanied by tinnitus, which will cause ischemia of cochlear structures and even neurogenic tinnitus.

Otolithiasis itself does not cause tinnitus, but it can cause some sub-health conditions of otolithiasis, which can induce tinnitus. There is no inevitable relationship between the two before, otolithiasis may cause tinnitus, and when the head is turned, dizziness will occur, and you should go to the hospital for surgery and reduction.

Under normal circumstances, patients do not have tinnitus, but some people do have tinnitus, especially neurological tinnitus is a product of sub-health, otolithiasis itself does not cause tinnitus, but it can cause some sub-health of otolithiasis, which can induce tinnitus.

1. Reset** Sit still for at least half a half.

Due to the reduction ** canalith displacement, the patient did not respond, Xu Suixian was more violent, and the vertigo reaction was more severe, and the patient could vomit 2, reduce ** 3, avoid lying flat within 3, avoid hairdressing and dental examination within the week, lie flat on the left side or lie on the right side, and the canalith has been reduced and can fall off again, resulting in re-dizziness 3, and the reduction ** adopts buckwheat skin pillow hard bed.

4. Reset ** to avoid strenuous movement.

In particular, low, shaking, jumping and other luck can easily lead to canalith falling off again, because vertigo can occur again, so before the recovery of otolithiasis, climbing, swimming and other dangerous luck should be avoided.

5. Reduction** To ensure sleep, rest, light diet, avoid smoking and alcohol, sleep enough, overwork, eat spicy, fat, fried and other hot foods, smoking and alcoholism are easy to induce otoliths to fall off again.

.Insufficient blood supply to the inner ear: due to arteriosclerosis, hypertension, diabetes and other factors, the glial membrane on the utricle cyst plaque is thinned due to nutritional disorders, and the canalith falls off and deposits in the semicircular canal.

2.Head trauma or ear surgery: after head trauma or accelerated head movement, such as whiplash injury, can cause the disease; Some middle ear surgeries, such as stapes surgery, can occur due to local pressure or stapes plate inserted into the vestibular window or avulsion of the canalith.

3.Ear diseases: middle ear mastoid infection, vestibular neuritis, viral labyrinthitis, Meni.

In general, otolithiasis is medically caused by brief episodes of vertigo and nystagmus caused by rapid head movement to a specific head position. Usually otoliths are attached to the membrane of otoliths due to some a**gens.

The main factors that cause otolithiasis are hereditary factors, endocrine disorders, bone labyrinth cyst development factors, autoimmune factors and other factors.

1. Hereditary factors There are more direct descendants of otosclerosis patients with the same disease, about 54% have a family history, some people think that it is autosomal dominant or recessive inheritance, and abnormal genes can be found in more than half of the cases.

2. Endocrine disorders This disease is more common in the onset of puberty, with a high incidence of women having a laughing disease, and can accelerate the progression of the disease during pregnancy, childbirth and menopause, which is believed to be related to hormone levels.

3. Factors for the development of bone labyrinth cysts Human beings have completed the development of bone labyrinth cysts at birth, but there is only a defect in the development and ossification process left in the endophytic cartilage layer of the anterior vestibular margin, which is called anterior window fissure. There are fibrous connective tissue bundles and cartilage tissue in the fissure, which can continue to exist or ossify after the Chengsheng Tomb and produce otosclerosis lesions, and the otosclerosis lesions seen in clinical and temporal bone pathology also start here.

4. Autoimmune factors and other Arslan and RIECI used histochemical staining to study the otosclerosis lesions, and found that in the active lesions, there were changes in mucopolysaccharide polymerization and the reduction and fracture of tissue fibers and collagen fibers, which were similar to pathological changes such as rheumatoid arthritis. In addition, it has been found that disorders of enzyme metabolism are responsible for the formation of stapes.

Otolithiasis refers to vertigo and nystagmus caused by a specific head position, which can be divided into primary and secondary, secondary mainly otitis media, Meniere's disease, migraine, trauma, etc., ** vestibular inhibitors can be used, such as phenagen and so on.

There are many reasons for this, it may be because you don't pay attention to your diet, and there is inflammation in the ear that is not taken care of in time, so it slowly changes to canalith symptoms.