Is it possible for diabetic ketosis to have an elevated base?

The common complications of diabetes include 1 kidney failure, 2 diabetic foot, 3 coronary heart disease, 4 stroke (cerebral infarction), 5 arteriosclerosis, 6 neuropathy, 7 retinopathy, 8 periodontitis, etc.

The three highs refer to high blood sugar, high blood lipids, and high blood pressure, which are most likely to cause cardiovascular complications.

Based on the results of your examination, it can be concluded that 1 your blood sugar is not well controlled (urine glucose +++ rapid blood sugar is slightly higher if it is 2 hours after a meal, and much higher if it is fasting blood sugar.

Dizziness, nausea and vomiting are typical precursors of cerebral infarction, and hospitalization** should be taken immediately to avoid cerebral infarction**.

According to the neurology department, the brain is examined, and the blood lipids (total cholesterol, high-density lipoprotein, etc., which are more demanding for diabetics, that is, the index is lower), blood glucose, glycosylated hemoglobin (reflecting the level of blood sugar control in the past three months).

After the examination, the cause of cerebral infarction is determined, and then the ** plan is determined.

Immediately find a neurologist and preferably hospitalized for a thorough examination.

Please refer to Qingdao Zesheng Toxoplasmosis Research Institute.

Because of severe electrolyte imbalances, hyposodium hypochloremia causes alkalosis, actively corrects electrolyte imbalances, refills saline, rehydrates and de-ketones, and adjusts blood glucose.

Diabetic ketoacidosis (DKA): is an acute complication of diabetes. It is acidosis triggered by a severe insulin deficiency caused by a sharp rise in blood glucose.

To put it simply, DKA is a serious occurrence of these two bad things at the same time: severe insulin deficiency and dehydration. Without enough insulin, in order to get energy, the body begins to break down fat, which breaks down to produce ketone bodies.

Normally, ketones are excreted in urine through the kidneys. But in DKA, ketones are produced too quickly, and the liver and kidneys don't have time to consume them, and the ketones are stored in the blood, making the blood acidic. At the same time, due to the lack of insulin, blood sugar continues to rise, but the body cannot use it.

Take a look: five chariots to calm blood sugar.

Does your father take acarbose again?

In diabetic ketoacidosis, the main harm of early alkali replacement is:

a.Secondary cerebral edema.

b.Secondary hypokalemia.

c.Secondary hypoglycemia.

d.Secondary hypocalcemia.

e.Secondary alkalosis.

Select: aSecondary cerebral edema.

Diabetic ketoacidosis is a symptom of pathological changes such as poor blood sugar control, large consumption of sweets, and obvious insulin insufficiency, resulting in a sharp increase in blood glucose, insufficient insulin secretion, increased blood ketones and ketonuria, dehydration, electrolyte imbalance, and metabolic acidosis.

Diabetic ketoacidosis may threaten the patient's life, causing coma, requiring aggressive hypoglycemia, correction of electrolyte acid-base imbalance, dehydration and other complications, and if it causes coma and impaired consciousness, vital signs need to be monitored.

Diabetic acidosis is the rapid decomposition of body fat leading to the rapid accumulation of acidic residues, that is, acidosis, alkali supplementation is used to neutralize acidic substances, and the body is strongly alkaline, then it is the opposite, it becomes alkalosis.

Severe deficiencies in insulin activity and inappropriate elevations of glycemic hormones cause disorders in the metabolism of sugar, fat, and protein, resulting in disorders in water, electrolytes, and acid-base balance. Hyperglycemia, ketosis, metabolic acidosis, and dehydration occur as a result.

Hello, how high is the patient's blood sugar now, is there a **?