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Arteriosclerosis is mostly caused by long-term hypertension, hyperlipidemia, obesity, smoking, drinking, or senile changes. Some elderly people are older, and they must be accompanied by arteriosclerosis, which is simply the aging of blood vessels. Basically, the elderly, all of them will have it.
The appearance of arteriosclerosis can be taken orally, anti-arteriosclerosis drugs, a prevention, to avoid the appearance of a cardiovascular and cerebrovascular disease. For example, light rippin, aspirin, atorvastatin, etc. It is necessary to control blood pressure, heart rate, blood lipids, blood sugar, and control these risk factors to delay the process of arteriosclerosis.
If there is anything uncomfortable, you should communicate with the doctor in time to avoid delaying the condition, I hope mine is helpful to you, thank you!
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Whether or not arteriosclerosis recovers depends on the severity of arteriosclerosis. For mild arteriosclerosis, if the patient's stenosis is less than 50%, there is no need to go through any intervention**. At this time, the main long-term medication is used to regulate the patient's blood lipids, and the general patients will not further develop arteriosclerosis after mediating the blood lipids.
A small number of patients can have a reversal of plaque, a reduction in the volume of plaque, and a reduction in the degree of arteriosclerosis, so it is possible to reduce arteriosclerosis after medication. However, arteriosclerosis can lead to further progression of the disease if risk factors persist in the risk layer.
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1. Arteriosclerosis is a non-inflammatory lesion of the artery, which can thicken the arterial wall, become better and harder, lose elasticity and narrow the lumen. Arteriosclerosis is a vascular disease that occurs with age, and its law usually occurs in adolescence, and it worsens and develops in old age. Aortic sclerosis is a common type of arteriosclerosis.
2. Aortic sclerosis ** measures:
a, the use of vasodilator drugs, such as prostaglandins.
b. Regulate blood lipids: on the basis of reasonable diet and appropriate exercise, correctly assess the risk of cardiovascular and cerebrovascular accidents in patients, and decide whether to carry out active lipid-lowering interventions, and oral statins can be used.
c, antiplatelet adhesion and aggregation: mainly including aspirin and clopidogrel.
d, thrombolytic drugs and anticoagulants: for intra-arterial thrombosis caused by lumen narrowing or obstruction, thrombolytic drugs can be used. Urokinase, fibrinosain, heparin, etc.
e, patients with arteriosclerosis can not drink alcohol and smoke, eat a low-salt and low-fat diet, have a regular life, and exercise appropriately.
Aortic sclerotic cardiac changes are herniated aortic segments on x-rays and are common in older people. If coronary arteriosclerosis is also present, it may be coronary heart disease, including asymptomatic myocardial ischemia, angina, myocardial infarction, heart failure and arrhythmia, and it is recommended to go to a regular hospital in time.
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Calcification of the heart's valves, such as the aortic valve, may be due to degenerative changes in old age, or rheumatic changes, or long-standing hemodynamic abnormalities. Calcification of the valve may affect the function of the valve, causing it to narrow or relocate, resulting in a series of abnormal changes. Only the calcification of the aortic valve has not affected the opening and closing of the valve, so it is not important at least for now, and it needs to be rechecked regularly to pay attention to the changes in the condition. >>>More
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Carotid arteriosclerosis, also known as carotid atherosclerosis, is a manifestation of systemic atherosclerosis in the carotid arteries, usually occurring in adolescence and gradually worsening with age. At present, it is believed to be closely related to the occurrence of ischemic stroke in the elderly. In the early stage of carotid arteriosclerosis, it is first manifested as intimart-media thickening, and then gradually forms atherosclerotic plaques, on the basis of which intraplaque hemorrhage, plaque rupture and shedding, mural thrombosis and secondary vascular stenosis occur, causing corresponding hemodynamic changes and leading to ischemic cerebrovascular events.