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Although rickets is a treatable disease, if it is not timely, the condition will worsen and even develop into physical deformities, which will affect the normal development of children in all aspects. The most common hazards are as follows:
1.Skeletal changes. Cranial deformity is more common in square heads, late teething, no teething for more than 10 months, lack of enamel in the teeth that erupt, and easy to caries; The fontanelle is enlarged and closed late.
Normal anterior fontanelles close after birth to one and a half years of age, but rickets do not close after one and a half years of age; The chest is chicken breast, pectus excavatum, etc., and the deformity of the thoracic cage will affect the respiratory function and reduce the lung capacity; Curvature of the spine, which can cause posterior or lateral curvature due to gravity and traction of the trunk; pelvic deformity, which can cause dystocia in adult female infants; The limbs are often bent, and the lower limbs form deformities such as knee valgus (called an "O" shaped leg) or knee inversion (called an "X" shaped leg). In severe cases, fractures may even occur.
2.The muscles are relaxed, the abdomen is distended, and umbilical hernias are prone to occur.
3.Neuropsychiatric symptoms may include night terrors, easy crying, and excessive sweating, especially in the first half of the night, when the person just sleeps, and has restless sleep.
As a result of the above conditions, the child is stunted, and both sitting and walking begin later than that of healthy children.
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My baby is now more than a week, and it is also rickets, and the doctor said that it will be good to supplement calcium, and it will not affect future development. The head was generous, and the cranial ribs turned out, and slowly recovered. Don't worry too much.
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I, female, had rickets, but recovered, now 26 years old, height, pay attention to the later diet matching, food calcium supplementation.
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In general, rickets does not affect mental development, and this disease mainly affects the normal development of human bones. Rickets is a chronic disease characterized by skeletal lesions caused by multifactorial calcium and phosphorus metabolism abnormalities and ossification disorders, which occur in the growth and development of children, among which vitamin D deficiency rickets <>
The most common, mainly built in infants and young children under two years of age, vitamin D deficiency rickets is caused by the lack of vitamin D in infants and young children caused by abnormal calcium and phosphorus metabolism, resulting in rapid growth of the long backbone of the limbs femoral cartilage plate, and other bone tissue calcification insufficiency, eventually leading to skeletal development deformities, and softening, in general, rickets only affects the child's bone development abnormalities, generally, does not affect intellectual development.
What are the dangers of rickets.
1. Cause skeletal deformity Rickets will be timely, and when the condition is severe, it can lead to skeletal deformity, mainly manifested as chicken breast, pectus excavatum, knee valgus, spinal curvature, etc., which affect the normal activities of patients, resulting in limited movement of the lower limbs and high disability rate.
2. Affect the psychology of rickets patients due to their face, specific disability, and limb dysfunction, and the patient has psychological damage, and is looked at strangely by others around him, and has excessive inferiority, depression, irritability, personality changes, and reluctance to contact others.
3. Induce other complications Rickets can lead to electrolytic balance disorders, and are prone to complications such as convulsions, dyspnea, and epileptiform seizures. When critically ill, it is life-threatening.
In general, rickets does not affect mental development, and this disease mainly affects the normal development of human bones. Rickets is a chronic disease characterized by skeletal lesions caused by multifactorial calcium and phosphorus metabolism abnormalities and ossification disorders, which occur in the growth and development of children, among which vitamin D deficiency rickets <>
The most common, mainly built in infants and young children under two years of age, vitamin D deficiency rickets is caused by the lack of vitamin D in infants and young children caused by abnormal calcium and phosphorus metabolism, resulting in rapid growth of the long backbone of the limbs femoral cartilage plate, and other bone tissue calcification insufficiency, eventually leading to skeletal development deformities, and softening, in general, rickets only affects the child's bone development abnormalities, generally, does not affect intellectual development.
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Rickets does not affect mental development, and rickets is generally a deficiency of vitamin D, which leads to the occurrence of low calcium.
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Rickets will not affect intellectual development, but it will affect physical development, so it is important to go to the hospital for a physical examination on time during pregnancy.
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If the baby has rickets, it will have a great impact on the baby's bone development, mental development, etc. Parents must pay attention to regular physical examinations and supplementation of trace elements.
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Rickets is caused by calcium deficiency, as long as it is supplemented in time, there is no problem.
During pregnancy, especially in the later stage, it is important to drink more milk, eat more fruits, eat more vegetables, and also eat some calcium tablets appropriately. Children can drink some cod liver oil from birth to 2 months, and at 6 months, they can drink Longmu Zhuanggu Powder, which has a good effect.
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To be precise, it should be a malnutrition disease caused by vitamin D deficiency.
Its full name is nutritional vitamin D deficiency rickets.
It is due to vitamin D deficiency, the intestinal absorption of calcium is reduced, and the body causes damage to the bones in order to maintain blood calcium concentrations.
Calcium supplementation alone is ineffective.
So it has nothing to do with heredity.
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Don't worry Now There are two kinds of rickets.
One is simple calcium deficiency, that is, insufficient calcium intake, or poor absorption. There are also cases where vitamin D intake is insufficient (e.g., eating too many foods can affect calcium absorption). Proper sun exposure can synthesize vitamin D) which is not hereditary.
The other is antivitamin D rickets (or familial antivitamin D rickets), which is inherited. (The information on this disease is available in the biology textbook of the second year of high school, and it is also available on the Internet, you can check it).
It is accompanied by X dominant genetic disease The disease-causing gene in men can only be passed on to daughters, that is, 50% of daughters are diseased, and 100% of sons are not diseased.
If neither of your parents has rickets, then you can rest assured that you are simply calcium deficient.
If one of your parents has rickets, there is a good chance that it is hereditary.
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I'm just here to tell you that it's not inherited.
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Rickets can be divided into the initial stage, the radical stage, the recovery period, and the sequelae stage. Generally, children over 2 years old who suffer from rickets have entered the sequelae stage, and the most common is children after 3 years old.
What is the sequelae of rickets?
The sequelae of rickets refer to the disappearance of the symptoms of rickets, the X-ray and blood biochemical tests are normal, and the skeletal changes do not progress, leaving only skeletal deformities of varying degrees. The most common sequelae are: costal margin valgus, chicken breast, pectus excavatum, "O" shaped legs, "X" shaped legs, etc.
Ways to correct the sequelae of rickets.
The skeletal deformities formed in the sequelae period cannot be reversed, and the key is to prevent them from continuing to develop. After the child enters the sequelae stage, there is no longer a need to add additional vitamin D and calcium, mainly through strengthening physical exercise to improve the deformed bones, without affecting the child's growth and development. Chicken breast refers to the obvious anterior protrusion of the sternum in children; In addition to eating more calcium-containing foods in the diet, children with chicken breast should do more exercises to expand the breast, such as passive and active exercises to expand the breast, swimming, etc., as long as they insist on exercising, with the increase of the child's age, the symptoms of chicken breast will be gradually relieved.
Children with "o" legs and "X" legs can be massaged with medial and lateral muscles to strengthen muscles, or wear appropriate orthopedic shoes and lower limb orthopedic sleeves. Severe deformities can also be corrected by surgery.
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Vitamin D deficiency, leading to calcium malabsorption! Vitamin D & Calcium!
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Get more sunlight, and also supplement calcium under the guidance of a doctor.
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First of all, I want to tell you that rickets is a relatively common disease caused by a lack of calcium and vitamin D in the human body, so it will not be inherited.
If a person has a very serious rickets, it is necessary to go to the hospital for a check-up to see what is causing it, and sometimes they have a special disease and inherit it to the next generation, which will also lead to insufficient absorption of calcium in the next generation, resulting in rickets.
In general, rickets can be seen when the child is three years old, so as a parent, it is necessary to observe whether the child has this tendency and actively supplement the child's nutrition.
However, there are some acquired rickets because of their own digestive system diseases that cause the human body to return to the body to absorb calcium normally, so it is necessary to actively cooperate**. Stuffy.
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