To what extent can progressive muscular dystrophy be controlled? How to take care of your body?

Progressive muscular dystrophy generally has a long incubation period and is not easy to detect, and when a patient finds out that he has this progressive muscular dystrophy, he or she already has a loss of muscle function, so it is a terrible disease. At present, in terms of scientific and technological means and medical means, there is no complete specific drug for this disease for the time being, but according to research, there is no problem at all for improving and restoring muscle function through drugs.

How to recuperate: 1. Physical exercise, massage to increase activity, promote blood circulation, prevent muscle atrophy, but should be moderate, not overworked. 2. Keep clean and quiet, pay attention to moisture and cold, and actively prevent complications such as respiratory tract infection.

3. Actively fight against the disease, appropriate recreational activities, promote patients to establish an optimistic and cheerful mood, and establish the confidence to overcome the disease with strong perseverance. 4. The diet should be light and nutritious, avoid eating or eating less greasy and thick overheat, wounding fluid and damaging the spleen and stomach, and eat more fish, eggs, chicken, lean pork, etc., but not too much, so as not to damage the spleen and stomach. 5. Maintain an optimistic and happy mood.

Progressive muscular dystrophy is a type of muscle degenerative disease caused by genetic defects, with progressive muscle weakness and atrophy as the main clinical manifestations.

Muscular dystrophy has a poor prognosis in most cases and can eventually lead to disability and death. At present, there is no **method, mainly symptomatic and supportive**. Appropriate training, timely application of braces to support the child's limbs, as far as possible to maintain and prolong the child's ability to walk independently.

Low-dose corticosteroids can lower creatine phosphosaryosase levels but do not stop the progression of the disease. Gene substitution** and small molecule** methods such as antisense oligonucleotide-induced exon skipping are still experimental and are expected to improve disease outcomes in the future.

The use of traditional Chinese medicine can have good results.

In addition to exploratory genes**, there have been three measures of Duchenne muscular dystrophy internationally, including glucocorticoids, exon skipping**, and no mutation inhibition.

Glucocorticoids are the most commonly used drugs for patients with Duchenne muscular dystrophy in Europe and the United States. Doctors generally recommend the use of defukol to help slow down the deterioration of muscle function. According to several studies, the use of defukol helps to maintain the stability of the heart and lungs in Duchenne muscular dystrophy and reduces the probability of severe curvature of the spine.

Although there is currently no Difukote listed in Chinese mainland, the Macau region has introduced Spanish Defcote. It is understood that DEFal was introduced by the Macau Five Continents Pharmaceutical International ****** and will be officially launched in Macao in October this year.

Exon skipping is a type of muscle malfunction that skips mutated genes to produce shorter, partially functional, malfunction proteins. Due to the presence of a mutant gene in one or more exons (part of a gene) in patients with DMD, the mutant gene is deleted. This can lead to errors in the instructions for making dystrophin and prevent the body from producing protein.

Exon**, on the other hand, can help patients produce partially functional muscle dysproteins.

Summary. Muscular dystrophy is not always inherited from the mother, and only the mother with pseudohypertrophic progressive muscular dystrophy is the carrier of the inherited gene. In boys, in particular, the disease manifests as weakness, atrophy, and impaired mobility of the proximal extremities.

Other ocular muscular dystrophy or limb-girdle muscular dystrophy is sometimes autosomal dominant or recessive, and some are sporadic, and it is not related to maternal inheritance.

Wait a minute, dear.

Hello, progressive muscular dystrophy is a kind of muscle resistance caused by genetic defects, the disease is currently not the first method, mainly symptomatic and supportive, and with appropriate training.

Is progressive muscular dystrophy only carried by women?

How muscular dystrophy is caused.

Myotrophic dystrophy is not necessarily inherited from the mother, only the mother of pseudohypertrophic progressive muscular rubber malnutrition is the carrier of the gene. In boys, in particular, the disease manifests as weakness, atrophy, and impaired mobility of the proximal extremities. Other ocular muscular dystrophy or limb-girdle muscular dystrophy is sometimes autosomal dominant or recessive, and some are sporadic, and it is not related to maternal inheritance.

Genetotrophic malasteria is a familial disorder in which patients present with progressive atrophy of muscles and often atrophy of limb muscles.

Is progressive muscular dystrophy only carried by women?

Progressive muscular dystrophy is not only carried by women.

Will the man carry it?

Yes. Can it be detected in the early stages of pregnancy?

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Progressive muscular dystrophy can be detected in the fetus during pregnancy, and you have to do an amniocentesis or cord blood puncture at 18 to 24 weeks. Please balance genetic testing. It is recommended that you go to a qualified hospital prenatal diagnosis center for regular prenatal checkups.

Oooh thanks. Hmmm! We're glad we could help! <>

Muscular dystrophy is divided into congenital type and limb belt type, all of which are caused by genetic defects, patients usually manifest as limb muscle atrophy, walking with a limp, difficulty swallowing, drinking water, choking, coughing, etc., walking like a duck's demeanor, and severe will lead to motor impairment or even paralysis. This disease is more common in children aged 3-5 years, who walk later than children of the same age, walk unsteadily, and are often mistaken by parents for reasons such as calcium deficiency or weak constitution and are ignored, and the symptoms become more and more obvious as the child grows older.

How about muscular dystrophy? According to the principle of traditional Chinese medicine, first of all, we must check the basic physical condition of the patient, find the root cause of the disease, muscular dystrophy is mostly caused by liver and kidney deficiency or spleen and stomach disharmony and other visceral problems, traditional Chinese medicine muscular dystrophy is mainly to regulate the viscera, the use of traditional Chinese medicine with instruments to dredge the meridians, regulate the whole body qi and blood, so that the growth factors can repair damaged and necrotic muscle cells after reaching the lesion, remove the inhibitory molecules that inhibit muscle cells, and regulate the structural and functional protein gene expression of target cells. In this way, muscle cell necrosis caused by gene defects is repaired, and muscle fibers are replenished, so that muscle cells no longer die. After the growth factors reach the lesion, it can produce a large number of myocyte trophic factors and myocyte immunomodulatory factors, improve the biological activity of myocytes, and on this basis, cooperate with acupuncture point dredging and targeting**, strengthen the metabolism of local muscle cells, and replace the various morphological and functional abnormal myotubes formed when lacking anti-myotrophin and the irregular morphology or hypertrophic or split myocytes of the lesions, so as to restore the normal strength, structure and function of the muscles, and accelerate the recovery of the disease.

If it is found that the person or child has muscular dystrophy or aggravates the family genetic history of muscular dystrophy, it must be diagnosed as soon as possible, as soon as possible, if not in time, as the disease progresses, the symptoms of muscle weakness become more and more severe, and the child around 12 years old loses the ability to walk independently. After long-term bed rest, it is easy to develop bedsores, and due to respiratory muscle weakness or heart involvement, respiratory failure and heart failure may occur and die at the age of 20.

So far, there is no specific method, only symptomatic and supportive, such as increased nutrition, normal activities, and so on. Physical and orthopedic forms can prevent and improve spinal deformities and joint contractures in patients and are important for maintaining motor function. Because progressive muscular dystrophy is a disease of the muscles themselves, especially in patients with rapid progression, more strenuous exercise is generally discouraged to avoid aggravating the condition.

However, patients should be advised to exercise appropriately and not to stay in bed for long periods of time. For drugs**, you can choose ATP, creatinine, vitamin E, etc. Gene and stem cell transplantation** has not yet been shown to be effective.

Since there is no effective method for this disease, it is particularly important to detect carriers, carry out prenatal diagnosis, and induce abortion of diseased fetuses.

Actively fight against the disease, adhere to appropriate recreational activities, encourage patients to build an optimistic and cheerful mood, and build up the confidence to overcome the disease with strong perseverance.

The diet should be modest, the pathogenesis of impotence syndrome is closely related to the deficiency of spleen qi, so the adjustment of diet is more serious, not too hungry or too full, in a regular, modest, at the same time, all kinds of nutrition should be properly deployed, not partial eating.

Actively fight against the disease, adhere to appropriate recreational activities, encourage patients to build an optimistic and cheerful mood, and build up the confidence to overcome the disease with strong perseverance.

At present, there is no specific method, only through symptoms and support, to delay the course of the disease.

Symptomatic** and general support** including vitamin E, inosine, galantamine, adenosine triphosphate, nandrolone phenylpropionate, and traditional Chinese medicine.

Proper functional exercises, full passive movement of each joint, acupuncture, massage, massage, etc. can delay the occurrence of severe muscle weakness, muscle atrophy and joint contracture.

You can use traditional Chinese medicine to try the effect, and the effect is still very good!

It's best to eat and exercise more than you usually exercise.

You can go to the ** of Chinese medicine and drink Chinese medicine to recuperate.

Muscular dystrophy is caused by genetic defects, patients usually manifest as limb muscle atrophy, walking limp, difficulty swallowing, drinking water, choking, coughing, etc., walking like a duck, severe will lead to motor impairment and even paralysis.

The use of traditional Chinese medicine** works very well and you can**.

1. Drugs**.

**Commonly used medications for muscular dystrophy include prednisone, methylprednisolone, sarcosinoside injection, lysinoinositol vitamin B12 oral solution, and diclofenac extended-release tablets. These drugs can help people maintain muscle strength, reduce pain as much as possible, and slow the progression of lesions in some patients.

2. Surgery**.

Some patients with myotonic progressive muscular dystrophy may need surgery to correct contractures or spinal curvature and improve quality of life and respiratory function.

3. Physics**.

Patients can do more stretching exercises to keep their joints flexible. You can also do some aerobic exercises, such as walking, swimming, etc., to maintain the strength of the muscles and prevent muscle tissue atrophy and fatigue.

The above are some of the best ways to perform muscular dystrophy, I hope it will be helpful to patients. Patients with muscular dystrophy also need to choose more foods rich in protein, vitamins, minerals and other nutrients, such as beans and yogurt, and maintain a low-fat and low-salt diet. If the confirmed patient has severe symptoms such as dyspnea and slow heart rate, he must go to the hospital for a follow-up examination in time to avoid more serious consequences.

There is currently no specific method for this disease. Only symptomatic and general support** should be used, including vitamin E, inosine, galantamine, adenosine triphosphate, nandrolone phenylpropionate, and traditional Chinese medicine. Proper functional exercises, full passive movement of each joint, acupuncture, massage, massage, etc. can delay the occurrence of more serious muscle weakness, muscle atrophy and joint contracture.

Active prevention and ** respiratory infections are valuable in prolonging the survival of patients. It has been reported abroad that the use of corticosteroids as the first of DMD has a certain effect on improving patients' muscle strength and motor function and delaying the progression of the disease. However, the long-term use of this type of drug is large, and its long-term efficacy is great.

Further observation is needed.

Genes related to DMD** are currently limited to animal testing. Since the DYS gene is one of the largest genes ever discovered in humans, it is difficult to mediate the entry of full-length 14 kbcDNA into muscle cells. Historically, viral or non-viral transgenic systems have had inefficient transfer and other problems.

Recently, researchers at the University of Pittsburgh have constructed a smaller than miniature DYS gene, which can be loaded into adenovirus-associated viral vectors and introduced into MDX murine murine myocytes, and the transfer system can maintain the expression of the most significant DYS protein for a long time, which is the most striking progress in the DMD gene. Stem cells are progenitor cells that can differentiate into a variety of tissue cells, and it has been reported that intravenous injection of normal hematopoietic stem cells can rebuild hematopoietic function in MDX rats and partially restore the expression of DYS in affected muscle cells, so stem cell transplantation has also become another hot spot in DMD** research in recent years.