Is Kawasaki disease a major illness? How should Kawasaki disease be treated?

Kawasaki disease is a major disease, Kawasaki disease, also known as pediatric ** mucosal lymphadenopathy syndrome, is a relatively common acute self-limited vasculitis in childhood, mainly with systemic vasculitis in children as the main pathological diseaseIt is more common in infants and young children, but it can also occur in school-age children, and it has relatively strict diagnostic criteria.

Symptoms of Kawasaki disease, such as the child's feverAnd antibiotics that last more than five days have no effect. Because it is called ** mucosal lymph node syndrome, the child will have a rash or conjunctivitis, mucositis, etc., as well as bilateral bulbar conjunctival hyperemia, then redness, chapped lips, bayberry tongue, congestion of the oral mucosa. A generalized, polymorphic rash may occur 2 to 3 days after the onset of illness, but it is usually transient.

In addition, the child will have more obvious swollen lymph nodes in the neckIt is usually unilateral, may be bilateral, and there is no redness, swelling, suppuration, and pain. Children with Kawasaki disease may develop hard edema of the hands and feet and film-like peeling later in the course of disease progressionThe biggest impact of Kawasaki disease on children is the heart and coronary arteries.

Some children with typical Kawasaki disease may have coronary artery aneurysm or coronary artery stenosis, and may also have myocardial infarction, which has a greater impact on the child's quality of life in the futureTherefore, if the child has persistent fever, as well as the manifestation of ** mucosal lymph nodes, it is necessary to confirm it in time, and if Kawasaki disease is diagnosed, it is necessary to actively treat the child with gamma globulin to avoid affecting the coronary arteries.

The first method of Kawasaki disease once the diagnosis of Kawasaki disease is confirmedIt must be done in the case of hospitalization**, and Kawasaki disease cannot be carried out at home**, which is to be emphasized. Hospitalization is performed after the diagnosis of Kawasaki disease**At the same time as hospitalization, you may need to do a lot of tests to help with it. First of all, it is necessary to clarify the trigger, whether there is an infection, and it may be necessary to do infection-related tests and examinations, and after it is clear that there is an infection, anti-infection** is the first step.

The second step is the specificity of KawasakiThere are two categories, one is the application of gamma globulin**, and the other is the application of aspirin, which is anti-inflammatory and anticoagulant**. Gamma globulin is the most specific form of Kawasaki diseaseIt is also the most effective method, and it is not possible to carry out a complete Kawasaki disease C-ball ** in the outpatient clinic or at home, so it is necessary to be hospitalized**, and gamma globulin ** is the most effective **method after the diagnosis of Kawasaki disease so far.

The second major type of Kawasaki disease ** is aspirin, aspirin is mainly used in two points, one is the anti-inflammatory effect of non-steroidal anti-inflammatory, because the pathology of Kawasaki disease is caused by systemic vasculitisVasculitis is also a non-infectious inflammation, and the use of aspirin as a non-steroidal anti-inflammatory drug for anti-inflammatory** is very effective, and aspirin also has a certain cooling effect and a certain anticoagulant effect. Therefore, these two categories are the most important and effective in Kawasaki disease.

This is a very serious disease, and its ** effect is not good, so it will not be added in the future. There are generally antiviral, anti-inflammatory and anti-psychoactive diseases, which can control inflammation and promote the body's resistance.

If the immunoglobulin** is ineffective, or is not resistant, glucocorticoids can be used early**, bed rest, adequate hydration, and the mucosal membrane can be kept clean.

Normally, there is no Kawasaki disease in the general critical illness clause of the insurance industry, but Kawasaki disease is very harmful to children, and some insurance product lines classify severe Kawasaki disease as a critical illness for young people. Glucocorticoids have a stronger immunosuppressive effect, which can inhibit the production of proinflammatory cytokines and block the inflammatory response. However, glucocorticoids are generally not used as the drug of choice for Kawasaki disease.

In general, in the insurance industry, there is no Kawasaki disease in the general critical illness clause, because Kawasaki disease is very harmful to children, and in some insurance product lines, severe Kawasaki disease is included in the scope of critical illness for teenagers. For those who have coronary artery dilation and aneurysm formation due to Kawasaki Hashizaki disease, and reach a certain level of severity, we provide critical illness protection to care for the healthy growth of children. The biggest harm of Kawasaki disease is to damage the coronary arteries of the heart, if there is no timely **, the coronary arteries dilate, produce aneurysms, may be life-threatening, even ** is also a potential risk factor for coronary heart disease in adulthood.

If the child has symptoms of a good face, it is important to consider Kawasaki disease, which is very simple, but the potential danger is very large, and the incidence has increased in recent years.

Disease Analysis: Opinion and Suggestion: Disease Analysis:

Kawasaki disease, also known as mucosal lymph node syndrome, is still unclear and is now mostly thought to be related to viral infection. It can occur in both infants and children.

Suggestions: The main symptoms of Kawasaki disease are common persistent fever, 5 11 days or more (2 weeks to 1 month in Lingsui), body temperature often reaches 39 or more, and antibiotics ** are ineffective. Bilateral conjunctival hyperemia, flushing, chapped or hemorrhagic lips are common, and bayberry-like tongue is seen.

There is hard edema in the hands, early flushing of the palms and soles of the feet, and characteristic patchy peeling of the toes after 10 days. There is also acute non-purulent transient cervical lymphadenopathy swelling, which is most significant in the previous neck, with a diameter of about 10 or more of the ridge, mostly unilateral, slightly tender, occurring within 3 days after fever and resolving spontaneously after a few days. Soon after the fever (about 1 4 days), a maculopapular rash or erythematous rash multiforme, occasionally a prickly rash, mostly on the trunk, but no herpes and crusts, subsided in about a week.

It often causes damage to the coronary arteries.