Can diabetes cause retinopathy? Is there a scientific basis for such a claim?

I think of course it is, because diabetes affects the liver, and a bad liver can lead to retinopathy.

Diabetes mellitus may cause retinopathy, which can be controlled by medication, diet and other methods to delay the development of the disease.

Diabetes can lead to retinopathy, this statement is scientifically based, as long as your blood sugar is not well controlled, then there will be complications, retinopathy is also a complication.

Hello, there is a correlation between diabetes and retinopathy!

The eye is a typical manifestation of microcirculatory disorders, which can cause a series of pathological changes, such as exudation, edema, vascular blockage, tissue and neovascularization, etc., and then cause intraocular hemorrhage, increased intraocular pressure (glaucoma), cataract and retinal detachment (retina refers to a thin film on the inner surface of the eyeball equivalent to the function of a photographic film, once it leaves its normal position, it will detach, and it will lose the function of transmitting photosensitive information) and blindness.

The occurrence of ocular complications is related to the duration of diabetes, and the occurrence of ocular complications occurs in 50% of patients with a history of 8 years. Almost all patients with a 20-year history of the disease have ocular complications, but the severity of the disease varies. It is also associated with good diabetes control.

Good blood sugar control can delay the onset and progression of ocular complications. If you have diabetes, it is important to have an ophthalmologist examine the fundus (i.e., examine the inside of the eye through the pupil) and continue to have regular check-ups in the future. In this way, we can achieve early detection and early **, and the effect is good.

Hello: Diabetes mellitus is a very harmful systemic disease, which is a disorder of glucose metabolism caused by pancreatic secretion dysfunction. There are a variety of ocular complications that can arise due to elevated blood sugar, the most common ones are:

Abnormal refractive changes in the eye: Diabetic patients will reduce the ion concentration in the aqueous humor due to the sudden increase in blood sugar, and the increase in the convexity of the lens due to excessive inhalation of water can lead to myopic refractive error, on the contrary, when the blood sugar decreases, the lens will lose water and hyperopic refractive error will occur, and it is often accompanied by obvious astigmatism. Therefore, when diabetic patients have sudden vision loss, they should undergo ophthalmometric refraction.

Diabetic cataract: Diabetic patients often have opacity of the lens, and the cataract complicated by juvenile diabetic patients is generally called true diabetic cataract. Diabetes also promotes the development of senile cataracts and accelerates their maturation.

Diabetic optic encephalopathy: It is the most common and serious diabetic eye complication, and is now one of the important diseases that cause blindness in the world. Diabetic optic reticitis often occurs in patients over the age of 40 who have had diabetes for a long time, and repeated vitreous hemorrhage and neoangiogenesis can lead to proliferative opticitis, resulting in severe vision loss and even blindness.

Paralytic strabismus: paralytic strabismus is common in patients with diabetes and is thought to be related to polyneuritis caused by diabetes. The main manifestations are sudden appearance of visual pairing, limited eye movement, vertigo and unsteady gait.

Other diabetic eye complications include uveitis, optic nerve optic meningitis, and neovascular glaucoma. For diabetic ocular complications**, blood sugar should be controlled first. Refractive anomaly vision loss caused by sudden changes in blood glucose usually resolves spontaneously after blood glucose stabilizes, and if vision does not recover after glycemic control, it should be corrected with glasses.

Patients with diabetes mellitus complicated by cataract should be treated with local intraocular infusions, catarrin, intracatual arrest, etc., and intraocular lens implantation should be done when the cataract matures. Diabetic optic membrane lesions are chronic progressive eye diseases, which eventually lead to severe vision loss and even blindness in patients. The best principle of diabetic paralytic strabismus is to control blood sugar first, and at the same time combine a large number of vitamins, neurotrophic agents and other drugs.

Diabetes mellitus may cause retinopathy, and may also cause diabetic foot and cerebral thrombotic complications, which will have a great impact on the patient's health. Therefore, the occurrence of the disease can be controlled through drugs, diet and other methods, delay the development of the disease course, and avoid the disease from continuing to be serious. Diabetes mellitus is a chronic disease that cannot be ***.

However, patients can delay the progression of the disease by taking drugs and traditional Chinese medicine, so as to effectively avoid the effects on the body and prevent other physical complications caused by the disease.

So, does diabetes cause retinopathy? What are the complications of this disease? Diabetes can cause retinopathy and, in severe cases, preretinal and intravitreal hemorrhages, which can lead to blindness.

Due to the occurrence of diabetes, it may lead to problems such as vascular lesions, which may lead to the phenomenon of lower limb lesions, which may cause complications such as diabetic foot in the patient's body. Diabetes mellitus is a disease in which glucose metabolism is abnormal. The occurrence of this disease may also cause problems such as abnormal protein and fat metabolism in the patient's body.

This can lead to an imbalance in the patient's internal environment, which can lead to coronary heart disease and other problems. Because the sugar in the blood of diabetic patients is relatively high, the blood will become more and more viscous, which will lead to more and more severe platelet aggregation, which will also cause the blood flow to slow down. So in this case, the patient's body is prone to complications such as cerebral thrombosis.

In the early stages of diabetes, the patient's body will have a relatively high level of albumin in the urine, and once this phenomenon occurs, it means that the glomerular function has irreversibly declined.

If the patient is not controlled in time, then it can lead to kidney failure and other complications. To sum up, there are many complications caused by diabetes, so patients with this disease must pay more attention to controlling sugar intake in daily life, and actively cooperate with doctors to delay the development of the disease. Patients should also pay more attention to appropriate physical exercise to promote the circulation and metabolism of the body and enhance personal physique.

Diabetic retinopathy (DR) is one of the most common microvascular complications of diabetes, which is caused by chronic progressive diabetes mellitus, which causes a series of fundus lesions, such as microangioma, hard exudate, cotton wool spots, neovascularization, vitreous proliferation, macular edema and even retinal detachment.

Not all diabetes will have this condition, only very severe diabetes patients will have a certain impact on the retina, so diabetics must pay attention to their diet.

This is not necessarily, some people have a stronger immune system and the symptoms are not particularly severe, so it will not cause retinopathy.

Diabetic retinopathy is one of the most serious complications of diabetes, and it is currently believed that long-term diabetes causes damage to retinal microcirculation, which will occlude retinal capillaries, lead to retinal ischemia, cause retinal edema and neovascularization. Most patients have the systemic symptoms of diabetes, and the main signs of retinopathy are: retinal microangioma, hemorrhage, oozing blood, venous bead-like changes, neoproliferative lesions, vitreous hemorrhage and traction retinal detachment, and in severe cases, blindness.

Diabetic retinopathy generally has no obvious symptoms in the early stage, once it is found that there is vision loss, visual distortion and other conditions should be paid attention to, retinopathy is often difficult to improve, so timely control of blood sugar is the most important part of the prevention and treatment of diabetic retinopathy.

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Japanese experts believe that diabetic retinopathy progresses in this way.

The occurrence and development of diabetic retinopathy can be divided into the following stages.

Non-proliferative phase. This is the first stage in the development of retinopathy. If diabetic patients do not control their blood sugar well, the retinal blood vessels will not flow smoothly under the influence of long-term hyperglycemia, and it is easy to form small tumor-like protrusions (capillary hemangiomas) on the tube wall. When these capillary hemangiomas break, they cause punctate or small patches of bleeding.

When the blood oozing out of the capillaries is absorbed, small white spots remain on the walls of the blood vessels. However, these pathological changes are subtle and not enough to affect vision.

At this stage, if attention is paid to blood sugar control in time and action is taken, there is hope to prevent the further progression of retinopathy.

Diabetic retinopathy (DR) is one of the most common microvascular complications of diabetes. In addition, diabetic retinopathy develops early and severe in type 1 diabetes, and retinopathy occurs later in type 2 diabetes.

There is an important retina like a camera film in the human eye, and many blood vessels are distributed on the retina, and diabetes makes the retinal blood vessels in a high-sugar environment for a long time, thus becoming fragile, just like a cracked water pipe, which is easy to leak and bleed, and becomes diabetic retinopathy. The lesion can cause retinal microangioma formation, hemorrhage, exudate, and vitreous hemorrhage. Neovascularization and traction retinal detachment.

Diagnosis (1) medical history: a detailed medical history is crucial, in addition to whether there are polydipsia, polyphagia, polyuria and weight loss and other typical manifestations of diabetes, we should also pay attention to understand the course of diabetes, the longer the course of the disease, the higher the incidence of diabetic retinopathy, the more serious, especially the discovery time of some diabetes does not represent the real disease time, because the systemic symptoms are not obvious, the actual course of diabetes is often found for a long time, blood glucose and urine glucose test is an important basis for understanding the degree of diabetes control.

2) Fundus examination: fundus examination is the main means of diagnosis, microaneurysm and/or small hemorrhage is always the earliest and more accurate signs of retinopathy, with yellowish-white waxy hard exudative spots, indicating that the vascular system is dysfunctional, permeability increases, blood components escape, and white soft exudation indicates severe microcirculation disorder, serious vascular destruction, there is no neovascularization at this stage, so it is called simple lesion, with the development of the disease, At this stage, multiple focal or extensive retinal hypoperfusion indicates that neovascularization will soon occur, and from the occurrence of neovascularization, that is, entering the proliferative phase, indicating that the retinal circulation can no longer compensate for tissue hypoxia.

3) Special examination: Before the lesion of the fundus, there have been some subclinical changes, such as abnormal fluorescence morphology, retinal electrophysiology and visual contrast sensitivity, etc., which have reference value for its early diagnosis, and various special manifestations of fundus fluorescein angiography are of great significance for the diagnosis and staging of the disease during the progression of the lesion.

Among the causes of aggravation of diabetic retinopathy, smoking, high blood pressure, alcohol consumption, and pregnancy can aggravate the lesions.

High and low blood sugar can worsen diabetic retinopathy and increase the risk of bleeding.

Blood sugar changes during pregnancy, and retinopathy will not develop if the medication is adjusted according to the changes in blood sugar and the blood sugar is well controlled.

Blood sugar, blood pressure, and blood lipids are all factors that add importance to omentopathy. Because diabetes is a vascular lesion, hypertension and hyperlipidemia will aggravate vascular lesions.

Both short-term and long-term glycemic control is important. The first is to control fasting blood sugar well, and the second is to control blood sugar after meals and at night and keep it stable. Third, blood pressure should be well controlled and stable. If the above three points are not well controlled, they will aggravate diabetic retinopathy.