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It is a germ that cannot be killed by existing antibiotics!
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Diseases caused by germs were no longer a deadly threat to humans, and antibiotics** were effective in every infectious disease, but this was before antibiotics were misused. Every year, 50% of antibiotics in the world are abused, and the proportion in China is even close to 80%. It is precisely because of the abuse of drugs that the germs quickly adapt to the antibiotic environment, and various superbugs are born one after another.
In the past, a patient could survive with a few dozen units of penicillin, but now millions of units of penicillin are ineffective for the same condition. Antibiotics cannot be controlled due to infections caused by drug-resistant bacteria, which eventually leads to the death of the patient. In the 60s, about 7 million people died each year from infectious diseases worldwide, and this number rose to 20 million at the beginning of this century.
The number of deaths from sepsis has risen by 89 per cent, with the majority dying from difficulties with medication caused by superbugs. Efforts have been made to find a new drug to defeat the superbug, but it has not worked. Moreover, as the world gradually reaches a consensus on the misuse of antibiotics, the status and efficacy of antibiotics are questioned and strictly regulated.
While the germs spread, the research and development of antibiotics gradually stalled. Losing the once powerful antibiotics, people began to look for inspiration to fight diseases from the simple treatment methods of the past. Finding a healthy and natural ** that uses human autoimmunity to defend against the attack of superbugs has become a new consensus of many people about disease.
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The root causes of superbugs are genetics and variation.
Organisms will pass on most of their traits to the next generation when they reproduce, but the next generation will have some variations. These variations mainly refer to changes in genetic material. Variation is random and non-directional (i.e., good or bad).
The environment makes natural selection for these offspring. After the survival of the fittest, the offspring that are more suitable for the environment are selected.
The direct cause of superbugs is the heavy use of antibiotics in humans, which has screened out many bacteria with strong drug resistance. When one day, the bacteria become resistant to all antibiotics, the bacteria are superbugs. Because there is no medicine left to kill it.
The use of antibiotics in humans has accelerated the rate of mutation, which in turn has accelerated the creation of superbugs. Antimicrobial peptides are just one of the most effective drugs that have been developed. The speed at which new drugs are being developed is far less than the rate at which bacteria mutate.
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The misuse of antibiotics causes the virus to produce antibodies and mutate, which is beyond the scope of human medical treatment, and there may be other factors in the mutation process, such as climate warming.
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If it is not the misuse of antibiotics, it is a genetic mutation, but the probability of a natural genetic mutation is very low, unless molecular means are used.
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Superbugs refer to the abuse of antibiotics to make bacteria more and more resistant, so this kind of bacteria is collectively called super, this kind of superbug can cause thick sores and poisonous blisters in people, and even gradually make people's muscles necrosis. What's even more terrifying is that antibiotic drugs don't work against it, and the patient will have terrible inflammation due to the infection, high fever, convulsions, coma and finally death. The terrible thing about this germ is not its lethality to people, but its resistance to ordinary bactericidal drugs - antibiotics, for which people have almost no medicine, and most antibiotics do not work against it, and use it as if it is useless (plasmids have multiple resistance genes).
Superbugs also refer to methoxybenzenicillin-resistant Staphylococcus aureus MRSA, which is a type of bacteria that is difficult to diagnose and treat because it is difficult to kill it with general antibiotics. Once humans, especially patients with low immunity, are infected with this germ, it often causes complications such as sepsis and pneumonia, which is life-threatening and quite dangerous for mothers, the elderly and children. Superbugs are mainly transmitted through contact, and the infection is mainly caused by people with low resistance, and will not cause great harm to the general population.
The most important preventive measures are to pay attention to personal hygiene, especially correct hand washing, strengthen physical exercise, eat a reasonable diet, pay attention to rest, and improve the body's resistance. If you go to the hospital to visit a patient infected with VRE, you should follow the guidance of the relevant personnel in the hospital, do a good job of disinfection and isolation, and avoid contracting this disease due to the visit.
Several hospitals in Paris, France, have admitted a large number of patients infected with a "superbug". The reason why it is called "superbug" is because this vancomycin-resistant enterococci (VRE) has broken through the vancomycin defense line, the "last bastion" of human beings against bacterial infection. In the last two years, 313 people in France have developed VRE urethral or gastrointestinal infections, of which 3 have died because there is no cure.
VRE infection is mostly in patients over 60 years old, and the longer the infectious disease**, the more likely it is to develop VRE infection, which can occur in the community or in hospitals, and nosocomial infection is higher than community infection. VRE infection mostly occurs in severe hospitalized patients with cancer, liver cirrhosis, chronic nephritis, uremia, cerebral infarction, etc., mainly causing lung and abdominal infections, followed by blood, surgical wounds, and urinary tract infections. Notably, some cases develop drug-resistant staphylococci prior to VRE infection.
Experts speculate that it is possible that staphylococci transmit their resistance plasmid genes to enterococci, resulting in VRE.
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Superbugs have no specific symptoms.
Because different bacterial infections have different symptoms, and superbugs don't produce new diseases, they just don't respond to antibiotics anymore, so for example, what is the original symptom of this bacterial infection, and when it transforms into a superbug, it's still this symptom, it's just harder**.
As an aside, NDM-1, which is all the rage at this stage, and MRSA 10 years ago, etc., are the result of bacterial mutations.
NDM-1 is a new type of gene that can transcribe and translate enzymes that hydrolyze most antibiotics, which is why bacteria with this gene can become superbugs.
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Causes thick sores and poisonous blisters, and makes people's muscles necrotic.
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Superbugs are a type of antimicrobial-resistant bacteria.
This super germ can cause sores and poisonous blisters in people, and even gradually make people's muscles necrosis. What's even more terrifying is that the antibiotic Senrisu drug does not work on it, and the patient will cause terrible inflammation due to infection, high fever, convulsions, fainting and finally death. The terrible thing about this germ is not its lethality to people, but its resistance to ordinary bactericidal drugs - antibiotics, for which people have almost no medicine.
In 2010, the United Kingdom** broke out: a new superbug NDM-1 was found in South Asia, which is extremely resistant and can spread globally.
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