How to understand cerebellar atrophy and cerebral atrophy?

1. Cerebral atrophy is larger than cerebellar atrophy.

Cerebral atrophy is mainly a decrease in the volume of brain tissue, a decrease in the number of cells, and an enlargement of the ventricles and subarachnoid space, which can be divided into five types:

1.Cerebral atrophy with cerebral cortex atrophy as the main pathological change.

2.Cerebellar atrophy is predominantly cerebellar pathology.

3.Hereditary cerebral atrophy: it is divided into autosomal dominant and recessive inheritance, and is clinically divided into spinal cord type, spinocerebellar type, cerebellar type and peripheral type according to the pathological characteristics.

4.Localized cerebral atrophy: It is brain tissue degeneration caused by local brain tissue lesions or local cerebral malacia caused by surgery, which can produce localized atrophy in and around the lesion site, also known as some kind of encephalopathy or sequelae of brain injury.

5.Degenerative brain atrophy: It is a common and frequent senile disease with different degrees of atrophy and degeneration of the brain, cerebellum and olive bridge, brain dysfunction, neurological dysfunction and dementia as the main manifestations.

Cerebellar atrophy is generally broadly defined as localized cerebellar atrophy in brain atrophy.

Cerebellar atrophy is not a disease but a neuroimaging finding. It can be seen not only in some hereditary and degenerative diseases, but also in some acute courses such as the late stage of acute cerebellitis and some drug poisoning, and even in some clinically asymptomatic people, cerebellar atrophy can also be seen in imaging examinations, especially in the elderly. Common features are decreased cerebellar volume and widened sulci on neuroimaging.

It can be divided into localized and generalized cerebellar atrophy. Because the cerebellum is mainly involved in the regulation of somatic balance and muscle tone, patients with cerebellar atrophy often have symptoms such as gait instability, ataxia, and slurred speech.

The cerebellum is only a part of the brain, and if the cerebellum is atrophied, there may be a disorder in movement, and if the brain is atrophied, it will manifest as a disorder as a whole. Cerebellar atrophy is a type of cerebral atrophy, and the principle of cerebral atrophy is removal; Activate brain metabolic function and indirectly inhibit the progression of the disease; Activates brain cells that are in an inhibited, dormant state; Reduce the occurrence of various symptoms and complications associated with brain atrophy; Maintain residual brain function and improve quality of life. Since vascular disease is the culprit of cerebral atrophy, it is necessary to actively prevent and treat those diseases that affect vascular health, such as hypertension, diabetes, hyperlipidemia, arteriosclerosis, etc., and adjust lifestyle, improve nutritional structure, and correct bad lifestyle habits; The drug should be controlled, of course, the drug should be gradual and consistent.

Cerebral atrophy refers to the atrophy of brain tissue, which includes many parts, including the brain, brainstem, and cerebellum, and the brainstem can be subdivided into the midbrain, pons, and medulla. Cerebellar atrophy refers to a specific part of the cerebellum, that is, the atrophy of the cerebellum. When older people do imaging examinations, they will find that there is brain atrophy, which is the same as wrinkles when they are older, so there is no need to worry too much.

However, there are some diseases that may cause atrophy of some brain tissue and other brain tissues that are not synchronized with the patient's age, such as multiple system atrophy, which causes dizziness and unbalanced walking. During the examination, if the cerebellum is found to be more atrophied than other parts, it can be a sign of the disease. So, in general, cerebral atrophy actually includes cerebellar atrophy.

Patients with cerebellar atrophy will have symptoms of ataxia, the patient will have unsteady walking, unsteady standing, drunken gait, easy to fall, and may lead to significant memory loss, slowed reaction speed, and may also have speech difficulties. If the disease progresses further, patients may have symptoms such as movement disorders, facial and tongue paralysis, choking on water, difficulty breathing, and even hemiplegia, incontinence, and inability to take care of themselves.

Name: Mr. Xu.

Age: 43 years old.

Course of disease: It was diagnosed as cerebellar atrophy, and the results of genetic testing were genetic mutations, and the main symptoms: unsteady walking, hand tremor, dizziness, tinnitus, dry mouth and bitter mouth, slurred speech, choking cough, sweating, dreams, fear of cold, cold hands and feet, chest tightness and shortness of breath, leg soreness, calf cramps.

Previously**: I took vitamin B1, coenzyme Q10, and other drugs, but my condition never improved.

Now**: Brain Walking Soup**.

After a course of treatment: dizziness, tinnitus, cough improvement, excessive sweating, and improved sleep quality.

After three courses of treatment: hand tremor improved, walking more steadily, sweating, fatigue improved, chest tightness and shortness of breath, leg soreness, calf cramps improved.

After six courses of treatment: drinking water and choking, swallowing recovery, hand tremor, head tremor has been restored, sleep quality has been restored, lower limb muscle strength has been basically restored, walking has been steady, urine and urine situation has recovered, dizziness, tinnitus, cough recovery, chest tightness and shortness of breath, leg soreness, calf cramps, leg cold has recovered, and then continued to consolidate a course of treatment, the symptoms are basically none, and now there is no recurrence in the return visit.

Cerebellar atrophy, also known as spinocerebellar atrophy, is a neurological disease that runs in a family and has a 50% chance of inheriting the disease in their children as long as one of the parents is affected by the disease. In the later stages of cerebellar atrophy, patients with cerebellar atrophy may speak very clearly, be unable to control pitch, or even speech, write unrecognizable, and have difficulty swallowing. Unable to stand or even sit up, need to rely on a wheelchair to walk, or stay in bed, unable to take care of themselves.

If the brain or peripheral nerves are affected, the patient's intelligence is affected. In the end, the patient died of various complications.

Opinions and suggestions: timely use of scientific and reasonable methods, symptomatic, so as to control the patient's condition more quickly. At present, the minimally invasive targeted technology that is more respected in the medical field has achieved good results in the clinical practice of cerebellar atrophy, and it is hoped that it can also help the patient's condition.

Cerebellar ataxia is one of the more common diseases. The occurrence of the disease has brought a lot of impact to the patient. It can even make the patient lose the ability to take care of himself.

Changes in muscle tone can change from decreased lesion to spasticity, and the ataxic gait can change to spastic ataxia. Unsteady standing, leaning forward or swaying from side to side, more prominent when standing on tiptoe or heel, and easy to fall are often the most common symptoms in the early stage of the patient.

Name: Mr. Guan.

Age: 43 years old.

Course of disease: suffered from cerebellar ataxia for 4 years, main symptoms: dizziness, blurred vision, slurred speech, inaccurate hand grasping, small strength, unsteady legs, shaky walking, diarrhea, cold thighs and calves, poor sleep, dreaming, easy to wake up, sweating at night, hot hands and feet at night, constipation, burning sensation in urine, heart discomfort, palpitation, occasional tinnitus.

Often dry cough, insufficient gas, choking cough when drinking water, heavy salt taste like to drink water, heavy menstrual flow and blackness, easy temper tantrum, tongue with tooth marks, tongue coating cracks.

Previously**: After taking clonazepam, butylphthalide, methylcobalamin and other drugs, the condition was controlled, but *** was too large, but the condition never improved.

Now**: Brain Walking Soup**.

After a course of treatment: dizziness and cough improved slightly, sleep quality recovered, blurred vision, blurred ghosting improved.

After three courses of treatment: walking more steadily, insomnia and dreams better, urine and urine improved, and the feeling of cold legs and feet was better.

After six courses of treatment: walking steadily, drinking water and choking, difficulty in swallowing recovered, speaking clearly, dizziness was gone, the whole body muscle strength was basically restored, blurred vision was basically recovered, the legs and feet were cold, night sweats, heart, tinnitus improved, and then continued to consolidate a course of treatment, the symptoms were basically none, and now there is no recurrence in the return visit.

Suggestion: Patients with cerebellar ataxia should also maintain daily nutrition and calorie intake, and at the same time, patients with many sequelae of cerebral hemorrhage are prone to aphasia and inability to express their physical condition, and cerebellar ataxia occurs, resulting in a lack of nutrients in the patient's body and need to be ingested from food

Cerebral atrophy and cerebellar atrophy are mainly different parts of atrophy, one is the atrophy of the cerebral hemispheres, and the other is the atrophy of the cerebellar hemispheres. Secondly, the main clinical symptoms are different

The location and clinical symptoms are different.

Cerebellar atrophy is mainly due to changes in the ventricles, sulci and gyrus of the cerebellum caused by various reasons, which is mainly manifested as the widening of the ventricles of the cerebellum and the deepening of the sulci of the cerebellum and the thinning of the gyrus, which is manifested as the appearance of a cavity in the cerebellum, the space becomes larger, and then the cerebellar tissue becomes smaller, which is called cerebellar atrophy.

Usually it can be seen in congenital factors, one is called cerebellar ataxia, which is related to genes and causes cerebellar atrophy. Acquired factors are called neurodegenerative diseases, which can also manifest as cerebellar atrophy. Cerebellar atrophy causes ataxia symptoms such as drunken gait, nystagmus, poetic speech, and impaired motor coordination, including the inability to counterfinger and decreased muscle tone.

Mild symptoms of brain atrophy mainly include the following symptoms:

1. The decline of motor function is mainly manifested in motor dysfunction, such as unsteady and uncoordinated walking.

Second, memory loss, poor calculation ability, manifested by near memory loss, and third, cognitive dysfunction, manifested by memory decline.

Cerebellar atrophy disease is a family dominant genetic disease, if the parents in the family have cases of cerebellar atrophy, the probability of children suffering from this kind of disease is very high, about 50% or more, this is also a very serious genetic disease, so we should pay attention to the condition.

Cerebellar atrophy, also known as spinocerebellar atrophy, commonly known as Alzheimer's disease, is a hereditary disease, it is clear that some people suffer from this disease, and their children have a 50% chance of inheriting this disease.

In fact, cerebellar atrophy is a geriatric disease, and in general, it is unlikely that a child will have the disease. According to medical cases, this disease is considered to be constituted by cerebrovascular disease, poisoning or traumatic brain injury. Therefore, it is of great significance to understand the cause and treat the symptoms.

Cerebellar atrophy is a slow progressive disease, cerebellar atrophy in the imaging examination found that the volume of the cerebellum becomes smaller, the sulcus is widened, can be divided into localized and extensive cerebellar atrophy, because the cerebellum is mainly involved in the regulation of somatic balance and muscle tone, so patients with cerebellar atrophy will have gait instability, ataxia, unclear speech symptoms, to give patients appropriate exercise and training, to let patients have appropriate physical exercise, such as regular walking and tai chi.

At the same time, in terms of language, patients should often sing and communicate with others, such as talking more about cross talk activities, which can improve cerebellar atrophy and continue to worsen.

Patients with cerebellar atrophy must be as early as possible, and it is recommended that patients take the method of physical plus traditional Chinese medicine to regulate from the root, and should pay attention to the diet, and should not eat spicy, spicy, pickled, and smoked foods. Eat more fresh foods containing high-quality protein, such as dairy, meat, soybeans and soy products.

Patients with cerebral atrophy, except for sweet fruits, can eat other fruits, but they must pay attention to not eating high-fat and high-calorie foods in their diet, because if they take high-fat and high-calorie foods for a long time, they will lead to an increase in blood lipids.

Patients with cerebellar atrophy should eat more high-quality protein, such as milk, chickens and ducks, fish, eggs, and soy products. Eat more vitamin-rich foods, such as fresh fruits rich in vitamin C, hawthorn, etc. Patients with cerebral atrophy must try to eat less flavoring agents such as monosodium glutamate in their diet at ordinary times.

Cerebellar atrophy is a chronic degenerative disease of the central nervous system with cerebellar ataxia and brainstem damage as the main clinical manifestations, which will have a serious impact on the daily life of patients.

According to each patient's individual symptoms and constitution, one-to-one symptomatic prescription can help patients with cerebellar atrophy ataxia control and delay the development of the disease, alleviate symptoms, reduce pain and improve the quality of life.