How is Parkinson s disease treated?

After a small broken step, it is necessary to carry out targeted training. People with Parkinson's disease have a smaller than normal stride and are an important factor in gait instability. Therefore, the first step for the patient is to take a larger stride when training.

In addition, brain pacemaker surgery** should be considered. Drugs are present, and surgery is the only effective method in this period. Not only does it reduce muscle tension in the limbs, but it also controls tremors.

After maintaining the overall state, the symptoms of small broken steps will naturally be controlled, because the body remains forward, the forward posture of swooping is getting faster and faster, and the two feet will tie each other, resulting in loss of balance and falling, this disease requires drug intervention to strengthen the protection of the general medopa ** is effective. Simply walking small steps, can not be diagnosed as Parkinson's disease, this phenomenon is necessary to actively take the elderly to the hospital for **, and then under the guidance of the doctor to control the condition with drugs. If you have Parkinson's disease, you must pay more attention to rest, drink plenty of warm water, urinate regularly, and keep the urinary tract open.

Small steps are common in Parkinson's disease, which is a common neurodegenerative disease in middle-aged and elderly people, and the main reason for inducing Parkinson's disease is the degeneration of nerve cells that partially control movement in brain areas, and the affected brain areas no longer produce enough dopamine, and dopamine is a chemical neurotransmitter that can work with acetylcholine to adjust and control human muscles, so patients may experience decreased muscle coordination. It is recommended to seek medical attention in time for relevant examinations, such as blood and cerebrospinal fluid examinations, cranial CT and MRI examinations, functional imaging tests, etc., to determine whether Parkinson's disease exists. If Parkinson's disease is diagnosed, promptly** is recommended to slow the progression of the disease.

Small broken steps in walking are mainly thought to be caused by Parkinson's disease.

The disease is mainly seen in the elderly and is a degenerative disease. At the onset of the disease, the secretion of dopamine in the basal ganglia area decreases, which causes increased muscle tone in the limbs, which can lead to stiffness of the limbs, and when walking, it will appear as a panicked gait, like small broken steps. After the onset of the disease, oral compound medopa is the main drug, and other drugs include trihexyphenidyl, amantadine, selegiline, pramipexole, etc.

Secondly, small broken steps in walking are also considered to be caused by secondary parkinsonism. The causes of this disease include cerebrovascular disease, intracranial infectious diseases, and traumatic brain injury. At onset, patients may have increased muscle tone in the limbs and small broken steps when walking.

February 27, 2015 - Looking at the old man in our family, he has been walking very awkwardly recently, taking small steps, as if he is afraid of falling, but there is no like, what is going on?. Can taking small broken steps be Parkinson's disease? Parkinson's disease is a typical table.

1 year ago - Gradually developed stiffness on one side of the body, abnormal gait, hunched back climbing stairs, walking with small broken steps, unable to lift hands to hold things, etc. Later, it was learned that all this was caused by abnormal muscle tone. Dystonia, on the other hand, is a major symptom of Parkinson's. .

Suspected Parkinson's disease, limbs do not have the strength to walk small steps, this may be relatively early, this can be controlled by taking trihexyphenidyl drugs, if the effect is better, then you can take it for a long time, you can.

The most common disease in the elderly is extrapyramidal lesions, of which Parkinson's disease is the most common. Parkinson's disease is common in older people, and it is mainly due to the reduction of substantia nigra cells in the brain, and the production of less dopamine later.

This is a symptomatic manifestation of Parkinson's postural behavior abnormality. It is manifested as the body leaning forward, the elbow and hip joints flexion, the initiation of difficulty and slowness when walking, the loss of coordinated swing of the upper limbs, and the small stride distance. If you take a step, you will rush forward in broken steps, and you will walk faster and faster, which is called "panic gait".

In addition, patients may experience symptoms such as rigidity and dyskinesia, often with tube-like rigidity or gear-like rigidity. Facial muscle rigidity reduces the blinking movements of the expression, resulting in a "mask face". The neck and trunk are stiff, forming a characteristic flexion posture.

Now it is slow to move, difficulty initiating, and decreased voluntary movements. Finger fine motor disorders such as writing, difficulty in unbuttoning, tying shoes, etc., difficulty swallowing, coughing when drinking, monotonous and low voice, be sure to go to the hospital for examination in time**.

Generally, the walking gait of patients with cerebral infarction is hemiplegic gait, that is, the limb on the paralyzed side drags or when the muscle tone increases in the later stage, a circle-like gait appears. Patients who walk with small broken steps need to be considered for the possibility of Parkinsonism.

Parkinsonism also has secondary to cerebrovascular disease, so we should be highly suspicious, and we should go to the hospital in time to let a specialist doctor evaluate whether the patient is parkinsonism. It is necessary to combine other clinical symptoms, such as the physical examination of the patient: to see if there is hypertonia, whether there is bradykinesia, clumsiness, whether there is resting tremor of the limbs, etc., to make a comprehensive judgment.

Parkinson's patients walk with small broken steps, which may be caused by dystonia, and there is no way to control the balance of their body, so they must reduce their steps to maintain balance. Parkinson's patients should go to the hospital for examination in time, and use drugs under the guidance of doctors**, and insist on using drugs to avoid further aggravation of the condition.

Gait disorder in Parkinson's patients is a very common clinical symptom, which is manifested as a panicked gait, that is, for example, a normal person wants to stand up from a chair and walks very freely, and quickly stands up. But Parkinson's patients can't do it, they often hesitate and pause when standing up like walking, it is very difficult to take a step, and once they start and can't stop their steps, it is as if there is an object in front of them to grab an object, showing the appearance of rushing forward, especially when turning, the patient is very difficult.

People live in a state of chronic "drug abuse", such as home renovation, poor ventilation of new cars, etc., which are the triggers for the increase in Parkinson's disease patients. Experts have found that regular exposure to gasoline, gasoline waste, organochlorine pesticides, paints, plastic resins, 1,2,3,4-tetrahydroisoquinoline (TIQ), as well as endogenous dopamine metabolites, 3,4-dihydroxyphenylacetaldehyde and other compounds can increase the risk of Parkinson's disease and parkinsonism.

This is because these compounds contain MPTP-like substances, which are not highly toxic in themselves, but they can be oxidized and metabolized by the B-type monoamine oxidase (MAO-B) of glial cells into highly toxic MPP+ (1-methyl-4-phenypyridine ion), which is ingested by dopaminergic neurons through the peripheral high-affinity dopamine uptake system, and aggregates within the microchondria, blocking the oxidative phosphorylation system of nicotinamide adenine dinucleotide (NADH) and interfering with adenosine triphosphate (ATP) synthesis. Poor celery further leads to degenerative necrosis of dopaminergic neurons. Dopamine is an inhibitory factor in the human body, its main role is to inhibit excessive muscle tone, ethionicone is an excitatory factor in the human body, they are in a state of balance, the human body will be healthy, and when dopamine secretion is reduced, acetylcholine, the excitatory factor is relatively rising, and the imbalance of the state of the two deficiency will increase muscle tone, muscle stiffness, and Parkinson's disease symptoms, leading to the occurrence of Parkinson's disease and Parkinson's syndrome.

The fundamental problem of the gait of Parkinson's disease patients is mainly "broken step, forward rush, and no swing arm", here, experts introduce several simple training methods that can be operated at home to help Parkinson's disease patients overcome the "panicked gait".

Visual cues to overcome broken steps.

People with Parkinson's disease have shorter than normal steps, which is an important factor in gait instability, so the first step to do is to make the patient take a larger step. You can use the method of visual cues to draw a clear horizontal line every 50-60 cm on the ground, so that the patient steps on a horizontal line every step, so that the patient can increase the step distance, and after repeated training for many times, you can form an unconscious habit, even if there is no longer a reminder of the horizontal line, you can also "take a big stride" to walk.

Auditory cues plus heel landing to overcome the forward rush.

The formation of the forward gait is mainly caused by two factors: first, the walking rhythm is not well controlled, and it is getting faster and faster; In the second walk, the center of gravity is forward, which in turn leads to the "chasing center of gravity". Therefore, the patient should first control the rhythm of walking, and the trainer can shout the password "one, two, one" or "left, right, left" by the side, and the patient walks strictly according to this rhythm, so as to avoid the forward gait of walking faster and faster; The second essentials is that when walking, we must remember that our normal gait is that the heel lands at the beginning, transitions to the outer edge of the foot, and then to the inner side of the foot, so the patient must land on the heel and overcome the forward shift of the center of gravity caused by the whole ball of the foot or even the forefoot landing, so that the center of gravity can be kept in the back foot when the forefoot lands, and the hindfoot support is the main thing to avoid "chasing the center of gravity".

You're going to go to the hospital to see this, we can't decide.