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Anonymous users2024-02-12Up to now, the specific ** that causes rheumatoid joints is not very clear, and the specific mechanism is not very clear, and it is still in the research stage, but now it is found that the following factors may have a certain correlation with the onset of rheumatoid joints.
First of all, immune disorders, which are the main mechanisms of rheumatoid arthritis, such as activated CD4T cells and human leukocyte antigens, are significantly related to the pathogenesis of rheumatoid joints. Some inflammatory factors, such as tumor necrosis factor interleukin I, interleukin VI, and interleukin VIII, promote the synovium to be in a state of chronic inflammation, which can also lead to the destruction of articular cartilage and bone.
Second, genetic correlation, current epidemiological investigations and genetic investigations have found that the incidence of rheumatoid joints is closely related to genetic factors.
Third, environmental factors, some infections, such as Mycoplasma bacteria and viral infections, can induce the onset of rheumatoid arthritis by activating lymphocytes and secreting inflammatory factors to produce autoantibodies.
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Anonymous users2024-02-11The onset of RA (rheumatoid) may be related to genetics, infections, sex hormones, etc. The pathology of RA-arthritis mainly includes synovial lining cell proliferation, interstitial infiltration of a large number of inflammatory cells, as well as the neovascularization of microvessels, the formation of vascular pannus, and the destruction of cartilage and bone tissue.
Rheumatoid arthritis (RA) is an unspecified chronic systemic disease with inflammatory synovitis. It is characterized by polyarticular, symmetrical, and aggressive joint inflammation of the facet joints of the hands and feet, often accompanied by extraarticular organ involvement and positive serum rheumatoid factor, which can lead to joint deformity and loss of function.
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Anonymous users2024-02-10Rheumatoid arthritis is an arthritis characterized by chronic inflammation of the synovial membrane of the joints, which is thought to be related to genetic, infectious, and immune factors. At present, the best method of rheumatoid is laser needle, we use a needle with a section to use the surgical effect to peel off adhesions, loosen muscles, restore local blood circulation, reduce the concentration of local pain-causing substances, and quickly relieve pain symptoms, and then introduce He-Ne laser irradiation, which plays a role in anti-inflammatory and analgesic and promotes tissue repair. You can choose according to your situation.
There are many factors, mainly considered to be heredity, infection, sex hormones, and diseases caused by the combined action of internal and external factors, which can cause a large number of inflammatory cell infiltration in the interstitium and the destruction of bone tissue. Once the disease is clinically diagnosed, it is necessary to have an early active ** synovial proliferation caused by autoimmune reactions, the formation of inflammatory cells and cartilage degeneration, the infection is mostly thought to be caused by group A streptococcus, and there may be genetic factors, and the joint deformation of severe cases affects daily life. There is currently no specific treatment for rheumatoid arthritis.
Non-pharmacological drugs may be used, such as nutrition and rest
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Anonymous users2024-02-09Rheumatoid can be caused by:
1. Infection. For many years, the infection has been suspected as the disease**. Many of the clinical features of the disease, such as fever, leukocytosis, regional lymphadenopathy, and redness, swelling, heat, and pain in the affected joints, are very similar to inflammation caused by infection.
Infection is thought to be the initiating factor, and there may be different infectious initiating factors in different patients. It has been reported that 50% and 80% of rheumatoid patients develop the disease after 2 to 4 weeks after suffering from recurrent pharyngitis, chronic tonsillitis, maxillary sinusitis, otitis media, cholecystitis, influenza and other streptococcal infections, so it is believed that the occurrence of this disease is related to streptococcal infection.
2. Endocrine disorders.
There is a lot of evidence that endocrine disorders play a role in the pathogenesis. According to the opinion of Canadian scholars. After the animals were injected with large amounts of auxin and deoxycorticosterone (which caused endocrine disorders in animals), joint lesions similar to rheumatoid arthritis developed.
3. Genetic factors.
The results of genealogical surveys have shown that the prevalence of rheumatoid in the family of rheumatoid patients is 2 to 10 times higher than that in the family of healthy people. A family survey of 2,113 rheumatoid patients in Japan found that 156 families had familial disease. The rate of rheumatoid factor positivity in close relatives is also 2 to 3 times higher than in healthy people.
In a study of 29 monozygotic twin pairs, Smith et al. found that two pairs had rheumatoid disease, and the other pair had rheumatoid disease at the age of 53 and 56 years even though they lived separately.
Kapsta et al. reported that a pair of monozygotic twins developed rheumatoid at the age of 3 and 5 years and had a mother with childhood-type rheumatoid arthritis. The fact that monozygotic twins or dizygotic twins born to patients with rheumatoid arthritis are susceptible to rheumatoid disease illustrates the role of genetic factors.
Fourth, autoimmunity.
Due to the progress of immunological experimental research, the autoimmune theory of the pathogenesis of rheumatoid arthritis has been generally accepted by most scholars. There are several points that support the autoimmune theory.
1. There are a large number of lymphocytes and plasma cells in the synovial tissue of the joint of patients with rheumatoid arthritis, reflecting the existence of an active local immune response;
2. The patient's synovial tissue can synthesize a large amount of immunoglobulins (including rheumatoid factor);
3. The joints (including synovial fluid) have complexes formed by immunoglobulins and complement. The decrease in complement concentration in the synovial fluid is consistent with the activity of the immune complex.
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Anonymous users2024-02-08Rheumatoid arthritis is an autoimmune disease that mainly erodes the synovial membrane of the joint, and the initial onset of the joint is morning stiffness, swelling, pain, and finally the joint may be abnormal, and the normal function is still lost. This disease is mainly related to autoimmunity, genetics, infections, smoking and other factors. But it also has a certain relationship with the cold and wet environment.
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Anonymous users2024-02-07Living in a damp place for a long time and taking cold water.
Rheumatoid is not complete**. Will do the opposite.
Rheumatoid arthritis is a very serious joint disease, and if this disease is particularly serious, it will completely drag down the family, because the mobility is limited and needs to be taken care of by the family.
Rheumatoid arthritis (RA), referred to as rheumatoid, is an autoimmune inflammatory disease with chronic, symmetrical, polysynovial arthritis and extra-articular pathologies (subcutaneous nodules, pericarditis, pleurisy, pneumonia, peripheral neuritis, etc.) as the main clinical manifestations. Its prominent clinical manifestations are anti-symmetrical polyarthritis, with joints such as hands, wrists, and feet most commonly affected; It presents with redness, swelling, heat, pain and dysfunction in the early stage, and may have varying degrees of stiffness and deformity in the joints, as well as bone and skeletal muscle atrophy, which is a disease with a high disabling rate. From the perspective of pathology, rheumatoid arthritis is a widespread inflammatory disease that mainly affects the synovial membrane of the joint (which can later affect the articular cartilage, bone tissue, joint ligaments, and tendons), followed by the connective tissues such as the serous membrane, heart, lungs, and eyes. >>>More
There is a recipe that can dispel wind and dampness, warm menstruation and relieve pain, which can be called the first way to dispel rheumatism. There are seven in the whole party, from the medical saint Zhang Zhongjing. >>>More
1. Eat less milk, goat's milk and other milk, peanuts, chocolate, millet, cheese, toffee and other foods containing tyrosine, phenylalanine and tryptophan, because they can produce arthritis mediators prostaglandins, leukotrienes, tyrosine kinase autoantibodies and anti-milk LGE antibodies, etc., which are easy to cause allergies and cause arthritis to aggravate or worsen. 2 Eat less fatty meat, high animal fat and high cholesterol foods, because they produce ketone, acids, arachidonic acid metabolites and inflammatory mediators, etc., which can inhibit the function of T lymphocytes, and easily cause and aggravate joint pain, swelling, bone decalcification and joint destruction3 Less sweets, because their sugars are easy to cause allergies, can aggravate the development of joint synovitis, and can easily cause joint swelling and pain to aggravate4 Drink less alcohol and coffee, poor and other beverages, and pay attention to avoid passive smoking, because it can aggravate the aggravation of arthritis.