What is the relationship between myocardial infarction and arrhythmia?

Myocardial infarction may be associated with arrhythmias, which are not necessarily associated with myocardial infarction.

What is myocardial infarction?

The arteries that provide blood to the heart are called coronary arteries, and if there is a blood clot in the coronary arteries and is blocked, then it will lead to insufficient blood supply to the heart, and the myocardial cells will be ischemia and hypoxia, which will eventually lead to regional myocardial infarction. The shape of myocardial infarction is mostly called a cone, and the extent involved is limited to the area of the myocardium where the blocked blood vessels are blocked.

What is arrhythmia?

A normal person's heart beats 60 to 100 times per minute, and if someone's heart beats abnormally in terms of number and rhythm, it is called an arrhythmia. On the heart, there are several parts that control the heart's beat, called the sinus node, the atrioventricular node, and the conduction fibers that connect these nodules. The sinus node is the most important part of the heart's conduction system, it is composed of autorhythm cells, these autorhythm cells move for about 60 100 times per minute, so the sinus node is the origin of the heart's beating, it is at the junction of the superior vena cava and the right atrium.

The atrioventricular node is located under the endocardium on the right atrial side of the lower atrial septum and controls the beating of the ventricles. Between the sinus node and the atrioventricular node, there are conduction fibers that connect them, and these fibers travel between the heart muscle cells and play a role in conveying the commands given by the sinus node and the atrioventricular node, thereby causing the heart to contract and relax, and then cause the heart to contract and relax.

After myocardial infarction, the conduction fibers existing between the myocardial cells lose their conduction effect, and they cannot carry out the orders given by the sinus node or the atrioventricular node. That is, the sinus node gives an order, the atrium contracts, and then the order is transmitted to the atrioventricular node, because the conduction fiber loses its effect, the atrioventricular node does not receive the order, then the ventricle will not contract, and the blood will fill in the ventricle, and in the long run, it will lead to ventricular hypertrophy and even heart failure.

Therefore, myocardial infarction may lead to arrhythmia, so if you feel that your body is abnormal, please seek medical attention immediately.

Myocardial infarction and arrhythmias are two different pathologies, but they are related, and in many cases, arrhythmias are caused by myocardial infarction.

First of all, the first point is that myocardial infarction is a phenomenon of severe ischemia in the heart, and the formation of a disease will cause death, and the second point is irregular heartbeat, which means that the heartbeat is abnormal, and then there will be some abnormal phenomena, such as cardiac arrest or palpitation, which is the relationship between the former and the latter.

Myocardial infarction predisposes to arrhythmias. Coronary artery ischemia after myocardial infarction causes ischemia and necrosis of myocardial cells, and the cell membrane of necrotic cells is unstable, which is easy to cause arrhythmia.

Arrhythmias must be eliminated in time to avoid becoming severe arrhythmias or even sudden death. In the case of ventricular fibrillation or sustained polymorphic ventricular tachycardia, asynchronous or synchronous DC defibrillation or cardioversion should be used as soon as possible, and synchronous DC cardioversion should be used as soon as possible when the efficacy of the ventricular tachycardia drug Yu He's is not satisfactory. Once premature ventricular contractions or ventricular palpation hypertachycardia are detected, lidocaine 50 100 mg intravenously is immediately given, repeated every 5 to 10 minutes, until the premature contractions disappear or the total amount has reached 300 mg, and the vertical intravenous infusion is maintained.

Amiodarone may be used for recurrent ventricular arrhythmias. Bradyarrhythmias can be treated with atropine intramuscularly or intravenously. Patients with second- or third-degree AV block who develop hemodynamic compromise with a temporary artificial pacemaker are indicated.

When supraventricular tachyarrhythmia cannot be controlled by drugs such as verapamil, diltiazem, metoprolol, digitalis preparations, and amiodarone**, synchronous direct current conversion can be used**.

After a myocardial infarction, some arrhythmias usually occur, but most arrhythmias do not cause myocardial infarction. Myocardial infarction is a persistent blood** disorder in the coronary arteries that causes certain myocardial cells to die due to ischemia and hypoxia. Arrhythmia refers to abnormal heart rhythm and conduction of the heart conduction system, such as common atrial premature contractions, premature ventricular contractions, atrial fibrillation and other abnormal heart rhythms, right bundle branch block, i-degree atrioventricular block, etc.

What is the relationship between arrhythmia and myocardial infarction? How to understand? Myocardial infarction and arrhythmias are related, but after acute myocardial infarction, some arrhythmias, such as early-onset ventricular and ventricular tachycardia, are prone to occur due to partial myocardial necrosis and myocardial membrane instability that results from death.

In the first days of acute myocardial infarction, the most common cause of mortality is sudden death due to a malignant arrhythmia for some reason, so patients with acute myocardial infarction should have an electrocardiogram monitored and should actively ** arrhythmias, such as ventricular premature contractions and ventricular tachycardia.

Myocardial infarction is caused by narrowing of the blood vessels of the heart and the detachment of plaque, which interrupts blood flow to the distal heart muscle. Patients with myocardial infarction have irregular heartbeats due to myocardial ischemia within 24 hours.

However, there are many causes of irregular heartbeats, and myocardial infarction as we know it is only a part of it. Arrhythmias can occur in a variety of heart diseases, high blood pressure, thyroid disease, and even non-organ diseases. On the other hand, if there is no coronary heart disease, arrhythmia does not induce coronary heart disease and myocardial infarction, but if there is a problem with the coronary arteries, angina may be induced if there is a persistent arrhythmia.

If angina pectoris is intermittent or persistent and not relieved, acute myocardial infarction may occur. The ** of arrhythmia is usually the first to determine whether there is **, to assess whether the arrhythmia affects the prognosis, whether it affects the quality of life, etc., and then decides whether active treatment is needed.

Arrhythmias are associated with myocardial infarction, and many arrhythmias are caused by myocardial infarction. Arrhythmias such as ventricular tachycardia or ventricular fibrillation. Causes of myocardial infarction include narrowing of the coronary arteries and high blood pressure.

Arrhythmia is just the loss of the beating rate of the heart, while myocardial infarction is the cause of myocardial infarction. Myocardial infarction, on the other hand, may be preceded by arrhythmias.

Arrhythmia and myocardial infarction are two different diseases, but they are closely related, arrhythmia refers to tachycardia or bradycardia, myocardial infarction is due to the narrowing of the blood vessels in the heart, resulting in insufficient blood supply to the distal myocardium.

Arrhythmias mainly include abnormal pacemakers and conduction abnormalities.

Due to the ischemia, injury, and necrosis of myocardial cells, myocardial infarction will inevitably affect its action potential, which will lead to the potential difference between these myocardial cells and between them and the surrounding healthy myocardial cells, which will become the existence of ectopic pacemakers.

Myocardial infarction is caused by severe narrowing or blockage of coronary arteries that cause ischemia, edema, injury, necrosis of myocardial cells, which also includes conduction cells, if the anterior descending artery is blocked, it causes ischemia or even necrosis of conduction cells, and if the right coronary artery is blocked, it is mostly due to the edema of myocardial cells that compresses the conduction system. Therefore, a decrease in ventricular rate is common in inferior myocardial infarction, and a temporary pacemaker needs to be implanted, and the heart rate can slowly recover after the acute phase of myocardial infarction has passed, but arrhythmias caused by anterior myocardial infarction usually require permanent pacemaker implantation.

Myocardial infarction, I'm not in this specialty.

It may be myocardial ischemia and hypoxia, myocardial cell injury or necrosis. The conduction function of the heart's electrical rays is impaired.

In fact, the cells of the heart are divided into somatic cells and conduction cells. Somatic cells are responsible for normal heart activity. Such as contraction and relaxation.

Conductive cells, such as the sinus node, cells of the atrial tract, are primarily responsible for the electrical activity of the heart. It's like an ECG is like a fiber optic cable, and a blood vessel is like a road.

Myocardial infarction is caused by acute occlusion of the coronary arteries, resulting in local necrosis of part of the conduction system due to severe and persistent ischemia. Therefore, the arrhythmia associated with acute myocardial infarction is caused by myocardial ischemia and necrosis of the conduction system. Many metabolites are released after myocardial avascular necrosis.

A large amount of potassium ions can form extracellular local hyperkalemia, which can make the local quiescent membrane potential close to the threshold potential, shorten the duration of the action potential, and form an injury current between the ischemic and non-ischemic areas. Due to the increase of potassium in the local necrotic area, conduction can be delayed and the refractory period can be shortened, which is conducive to the occurrence of agonistic reentry. In acute myocardial infarction, catecholamines in the blood increase, which excitates receptors, thereby improving the automaticity of pacemaker cells and causing tachyarrhythmias.

In addition, catecholamines can lower the threshold of ventricular fibrillation, causing the development of ventricular fibrillation. Acute myocardial infarction can cause severe impairment of cardiac function, and metabolic acidosis may occur, which lowers the threshold current of ventricular fibrillation. If respiratory ventilation is impaired, hypoxemia with respiratory alkalosis may occur.

It can change the distribution of ions inside and outside the pacemaker cell, reduce the extracellular liquid alkali reserve, and potassium and sodium ions enter the cell, enhance the automaticity of the pacemaker cell, and can cause ventricular and supraventricular tachyarrhythmia. In addition, myocardial ischemia can cause hyperarrhythmias, which in turn can cause tachyarrhythmias. Myocardial ischemia can reduce myocardial contractility and increase left ventricular terminal diastolic pressure, which in turn strengthens the automaticity of pacemaker cells, turns non-pacemaker cells into slow fibers with pacing performance, and can make the refractory period and conductivity of various parts of the myocardium inconsistent, thus causing arrhythmia.

Myocardial infarction often causes some arrhythmias, but arrhythmias often do not lead to myocardial infarction. Myocardial infarction is a persistent blood disorder in the coronary arteries, resulting in the necrosis of some myocardial cells due to ischemia and hypoxia. However, arrhythmias refer to abnormal rhythms of the heart's beating and abnormal conduction of the cardiac conduction system, such as common heart rhythm abnormalities such as atrial premature contractions, ventricular premature contractions, atrial fibrillation and right bundle branch block, atrioventricular block and other conduction system abnormalities.

According to the latest news I have learned, myocardial infarction has a certain correlation with arrhythmia, but after the occurrence of acute myocardial infarction, due to the instability of some myocardial necrosis and necrotic myocardial cell membranes, some arrhythmias, such as ventricular premature contractions and ventricular tachycardia, are easy to occur. The most common reason for the high mortality rate of most acute myocardial infarctions in the first few days is that malignant arrhythmias are prone to occur after myocardial infarction, so patients with acute myocardial infarction should be monitored electrocardiogram and actively manage arrhythmias such as premature ventricular contractions and ventricular tachycardia.

However, there are many causes of arrhythmias, and patients with myocardial infarction are only part of the cause. Arrhythmias can occur in a variety of heart diseases, high blood pressure, thyroid disease, and even non-organic diseases. On the other hand, if there is no coronary heart disease, arrhythmia does not induce coronary heart disease and myocardial infarction, but if there is a problem with the coronary arteries, angina can be induced if persistent tachyarrhythmia occurs.

If angina persists and does not resolve, acute myocardial infarction may occur. Arrhythmia is generally to determine whether there is a **, assess whether the arrhythmia has an impact on the prognosis, whether it affects the quality of life, etc., and then decide whether to be active**, myocardial infarction is also known as myocardial infarction, myocardial infarction refers to coronary artery occlusion, interruption of blood flow, severe continuous ischemia leading to partial myocardial necrosis.

Finally, as for the above question of how myocardial infarction is related to arrhythmia and what it manifests, I will analyze it here today.

Arrhythmia is one of the symptoms of myocardial infarction, and some headaches, vomiting, and angina may occur.

There is a correlation between myocardial infarction and arrhythmias, and angina may be induced if persistent rapid arrhythmias occur; If angina persists and does not resolve, an acute myocardial infarction may occur.

Myocardial infarction means that the heart has some steal arrest, at this time the blood clotting together will form such an infarct arrhythmia, that is, the heartbeat is erratic, sometimes fast and sometimes slow, this is arrhythmia, like when we are usually emotional, this aspect can be well expressed.