Atherosclerotic lesions occur predominantly:

Immune inflammation of the arteries after endothelial injury? (The mechanism is not very clear) - atherosclerosis.

It occurs mainly in the intima of the great middle artery. The main thing is the pressure it is subjected to by the impact of the blood. Although the causes and mechanisms of atherosclerosis are not well understood.

But all arteries are possible.

The carotid artery is the most common noninvasive test for the presence of atherosclerotic blood vessels in the artery (by ultrasound testing).

Atherosclerosis is characterized by non-inflammatory, degenerative and proliferative lesions in the arteries, resulting in thickening and hardening of the walls, loss of elasticity and narrowing of the lumen. The pathological changes of atherosclerosis mainly involve large elastic arteries of the systemic circulatory system (

For example, the aorta and the medium-sized elastic arteries (with the coronary arteries and cerebral arteries being the most affected), the lesions of the affected arteries start from the intima, and a variety of lesions exist at the same time, including local accumulation of lipids and complex sugars, hemorrhage and thrombosis, fibrous tissue hyperplasia and calcinosis, and gradual degeneration and calcification of the middle layer of the arteries. Modern cell and molecular biology techniques have shown that atherosclerotic lesions are characterized by smooth muscle cell proliferation, the formation of a large number of connective tissue matrix such as collagen fibers, elastic fibers and proteoglycans, and the accumulation of lipids inside and outside cells. Because the lipids that accumulate in the lining of the arteries have a yellow atherotic appearance, they are called atherosclerosis.

Answer]: C Atherosclerosis mainly affects the large and middle arteries, and the basic lesions are the accumulation of lipid collapse of the arterial intima and the accumulation of sedimentary mass, intimal focal fibrosis, and the formation of atheromatous plaques, which cause the wall of the tube to become stiff and the lumen to narrow, and cause a series of secondary lesions, especially in the heart, brain, kidney and other organs, which can cause ischemic changes.

a.Blood quarrel and empty cherry bolt formation.

b.Fiber deficit cap formation.

c.Foam cell formation.

d.Necrotic foci form.

e.Helcosis.

Correct answer: c

Answer]: A This question examines the knowledge points of pathology-cardiac paracanal system-atherosclerosis. Atherosclerosis is the paper deposition of the arterial intima with the collapse of the intimal focal, the formation of atheroma and oak, causing the wall to harden and narrow the lumen, causing ischemic changes in the corresponding organs, which are more common in the elderly, and the most common site is the large and middle artery (abdominal aorta).

Therefore, option A should be selected for this question.

Single-choice] Atherosclerosis occurs mainly in:

a.Thin and small arterioles.

b.Great and middle arteries.

c.Thin and small veins.

d.Large and middle veins.

Answer] B Analysis] Atherosclerosis is the most common disease of the cardiovascular system, characterized by the formation of atheromas or fibrous plaques in the lining of blood vessels, mainly affecting the posterior walls and branch openings of large and medium arteries.

Answer]: The process of the occurrence and development of atherosclerosis can be roughly divided into the following four stages: Striae is the early change of atherosclerosis.

To the naked eye, the surface of the arterial intima appears yellow markings 1 2 mm wide and of varying lengths, flat or slightly raised. Microscopically, lipid streaks are formed by the accumulation of a large number of foam cells containing lipoproteins in the cytoplasm under the arterial intima. As the lesion continues to develop, fibrous plaques gradually form.

To the naked eye, the inner membrane surface is scattered with irregularly raised fibrous plaques that appear yellow in the early stage. As the fibrous tissue grows and becomes vitreous, the plaque becomes porcelain-white. Microscopically, the surface layer of the lesion is a fibrous cap formed by a large number of collagen fibers, smooth muscle cells, a few elastic fibers and proteoglycans, and the collagen nuclei hail fibers can undergo vitreous transformation.

The lesion further develops into atheromatous plaques, visible to the naked eye, grayish-yellow plaques on the intimal surface of the arteries. On the incision surface, the luminal surface of the plaque is a white fibrous cap, and the deep tissue is necrotic and disintegrates, and the necrotic tissue is mixed with the lipid in the lesion to form a yellowish-white, viscous athero-like substance. Microscopically, the surface of the atheromatous plaque is a layer of fibrous caps, the deep layers are amorphous necrotic disintegrating substances, cholesterol crystals inside, granulation tissue and fibrous tissue at the bottom and edges, and a small amount of foam cell aggregation and lymphocyte infiltration.

Secondary lesions are secondary lesions that are based on fibrous plaques and atheromas: a Intraplaque hemorrhage; b Plaque rupture; c Thrombosis; d calcification; e Aneurysm formation.

Characteristics of atherosclerosis tard-sclerosis lesions ().

a.The wind is held in the wet body of the plum.

b.Great and medium arteriosclerosis.

c.Arteriosclerosis of arterioles.

d.Capillary sclerosis.

e.Hardening of small veins.

Correct answer: B

In atherosclerosis, which of the following lesions does not appear?

a.Lipid infiltration of the arterial intima forms lipid striaes.

b.Collagen fibers and vitreous changes in the arterial intima.

c.Calcifications may occur within atheromas.

d.Severe lesions may present with medial failure, smooth muscle atrophy, and elastic plate rupture at the site of severe lesions.

e.Fibrinoid necrosis occurs at the site of the lesion.

Correct answer: e

Answer]: B atherosclerosis occurs mainly in large arteries (such as aormomorphic fibrillation arteries) and middle digital arteries (coronary arteries, cerebral basilar arteries, renal arteries, and extremity arteries).