What is the term explanation for pulmonary encephalopathy?

Pulmonary encephalopathy (PE) is also known as emphysema.

Encephalopathy, carbon dioxide anesthesia, or hypercapnia, is caused by various chronic pulmonalmothoracic diseases associated with respiratory failure, resulting in hypoxemia.

and hypercapnia, a clinical syndrome of various neuropsychiatric symptoms.

a.Alveolar hypoventilation leads to brain dysfunction.

b.Dysfunction of cerebral function due to hypoventilation of the lungs.

c.Brain dysfunction due to respiratory failure.

d.Brain dysfunction caused by "CO2 anesthesia" is ruined.

e.Brain dysfunction due to hypoxia.

Correct Answer: Brain dysfunction due to respiratory failure.

Pulmonary encephalopathy is chronic bronchitis complicated by emphysema, cor pulmonale and pulmonary failure caused by brain tissue damage and cerebral circulatory disorders. The main manifestations are headache, dizziness, memory loss, and lack of energy.

The main cause of pulmonary encephalopathy in the elderly is chronic cor pulmonale, and the common causes are acute respiratory and pulmonary infections, severe bronchospasm, and sputum obstruction in the airways further reduces the already impaired pulmonary ventilation function.

The full name for cor pulmonale is chronic cor pulmonale. Its symptoms depend on the progression and exacerbation of the primary disease, and eventually develop into cor pulmonale. Cor pulmonale is a more common cardiovascular and cerebrovascular disease in life, this disease occurs more in many elderly people, if the patient has pulmonary heart disease, then there will be some more obvious symptoms, which can be diagnosed through these symptoms, and the symptoms of different patients are different.

Let's introduce how to judge whether you have pulmonary heart disease.

Coronary heart disease may be diagnosed if two or more diagnostic criteria are met

1.Cor pulmonale is divided into acute and chronic, and the diagnostic criteria are slightly different.

2.Chronic cor pulmonale: cough, sputum production, shortness of breath in the early stage, respiratory failure and other symptoms in the later stage, it is recommended to be prompt**.

3.Acute cor pulmonale: patients will have symptoms such as chest pain, suffocation, and dyspnea, and severe patients may have symptoms such as shock, and should seek medical attention urgently.

For cor pulmonale, the principle is mainly to reduce the patient's symptoms, improve the patient's quality of life and activity endurance, reduce the number of acute exacerbations, and improve the survival rate of the patient. Acute cor pulmonale and acute exacerbations of chronic cor pulmonale are usually in critical condition and require resuscitation. The remission period of chronic cor pulmonale is mainly through daily conditioning, medication**, etc.

Traditional Chinese medicine can be used**, licorice fine spicy decoction ** pulmonary heart disease effect is remarkable, through clinical application and continuous improvement, this prescription is not like Western medicine or traditional Chinese medicine as simple sterilization, so as to produce drug-resistant strains, so that pulmonary heart disease becomes a stubborn disease. On the one hand, it can inhibit the metabolism of pathogens, on the other hand, improve the function of cellular immunity and humoral immunity, phagocytosis of pathogens through the lymphokines of immunoglobulins, activate complement, neutralize toxins, fight infection, eliminate inflammation, and eliminate bacteria and viruses. At the same time, its medicinal ingredients can effectively restore and improve the immune function of the respiratory system, build a solid immune protective barrier, prevent reinfection, and also have the characteristics of safe and non-toxic, fast effect, low cost, and difficult to heal.

Abbreviated as "cor pulmonale". It refers to the increase of pulmonary circulatory resistance caused by the lesion of lung tissue or pulmonary artery and its ulnar branch, and the increase of right ventricular load, resulting in the enlargement of the right heart, with or without burial with congestive heart failure.

1.Blood gas analysis showed an increase in PAC2, an increase in carbon dioxide binding, an increase in the content of standard bicarbonate and residual alkali, and a decrease in blood pH.

2.Increased cerebrospinal fluid pressure, increased red blood cells, etc.

3.Electroencephalogram (EEG) shows diffuse chronic wave abnormalities of varying degrees and may have paroxysmal changes.

Mainly based on chronic lung disease with pulmonary failure; Clinical manifestations include impaired consciousness, neurological, psychiatric symptoms, and localized neurological signs; Pulmonary insufficiency and hypercapnia on blood gas analysis; Diagnosis is made after excluding neurological and psychiatric disorders caused by other causes.

1. Pulmonary encephalopathy is a symptom of insufficient oxygen supply caused by respiratory failure.

It is generally manifested as apathy, drowsiness, coma, etc.

Abnormal changes due to ischemia and hypoxia may occur within the skull.

2. When he wakes up, he may be holding his breath and waking up, so he will have a sense of impending death, so he will resist desperately, and his strength will be great.

3. Protein infusion is ineffective, because it is not caused by hypoproteinemia, oxygen inhalation should be effective, and it is better if it can be supported by a ventilator.

Pulmonary encephalopathy refers to brain dysfunction caused by respiratory failure. The pathogenesis is as follows: carbon dioxide directly dilates cerebral blood vessels, and hypoxia can also dilate cerebral blood vessels, thereby causing cerebral congestion; Hypoxia and acidosis damage the vascular endothelium, increasing its permeability, resulting in interstitial edema; Hypoxia reduces the production of ATP in brain cells, affects the function of NAA pump on brain cell membranes, causes intracellular water and sodium retention, and forms cerebral cell edema. Cerebrospinal fluid (CSF) pH decreases in respiratory failure, leading to intracellular acidosis, which can increase cerebral glutamate decarboxylase activity and increase the production of B aminobutyric acid, leading to central inhibition.

In short, due to the above factors, cerebral congestion, edema, increased intracranial pressure and even brain herniation are formed, coupled with central inhibition, resulting in severe brain dysfunction.