What are the symptoms of avian encephalomyelitis in pigeons?

Avian encephalomyelitis is an acute, highly contagious infectious disease caused by a virus that mainly affects the central nervous system of young chicks, with progressive ataxia, head and neck tremor, unsteady standing, paraparesis and incomplete paralysis of the limbs as the main symptoms, and non-purulent encephalitis as the main lesion. The disease has been shown to exist in most areas of commercial poultry farming. In recent years, there have been outbreaks of this disease in Heilongjiang, Guangdong and other places in China, and caused epidemics.

The disease spreads rapidly, and adult hens do not show clinical symptoms after infection except for a decrease in egg production, but the virus can be transmitted vertically to the next generation, and cause continuous morbidity and death of young chicks, which brings great threats to the poultry industry. Here are a few measures to prevent and treat avian encephalomyelitis in chickens.

Pigeons with typical symptoms should be immediately singled out and culled, burned or cull for deep burial in order to reduce the infection of the same flock.

Use peracetic acid to disinfect with pigeon spray, and appropriately raise the temperature of the brooding house, give a comfortable environment, and eliminate all kinds of adverse stress factors. Antibiotics are used to prevent secondary infections.

At the same time, vitamin E, vitamin B and selenium should be appropriately added to the feed, which can play a role in protecting nerve tissue and improving clinical symptoms.

At present, there is no effective drug for avian infectious encephalomyelitis. The attenuated vaccine has a certain pathogenicity to chicks and should not be used for small day-old pigeonsHowever, for breeders, vaccination against infectious encephalomyelitis before the start of production allows them to develop immunity and pass on maternal antibodies through the eggs to the next generation, which can be retained in the chicks until 6 to 8 weeks of age.

Maternal antibodies protect chicks from viral infection during the critical 2-3 weeks of age.

Avian encephalomyelitis virus is highly contagious and can be transmitted by contact or through eggs. For this reason, the breeding flock is vaccinated with a weakened vaccine during the growing season. More knowledge of pigeon breeding is in the pigeon racing world network "pigeon hall", pigeon fancier experience exchange "pigeon world forum".

Crooked head syndrome, which is an unhygienic bird and feed and pigeons that may be transmitted to other pigeons during the release process, must be isolated in the whole cage when this problem is found.

This is Newcastle disease, avian encephalosponditis is not contagious, and the incidence is very low, there will be no changes in muscles and eyes, the symptoms are mainly pumping, no ability to fly, there will be bleeding symptoms in the nose and mouth, I have seen a case in more than ten years. I persisted for half a month and finally hung up, I didn't understand it at first, and finally the results of the dissection and laboratory examination of the meninges.

Viral** can be used, starting at 1 day of age.

Prevention and control measures] 1 Strengthen the usual hygiene management The brooding room should be commonly disinfected with 100 poisons in an appropriate proportion with chicken spray, and the sick chicks and dead chicks that have been sick should be burned or buried deep in time to avoid spreading the virus. When this disease occurs in breeders, the eggs should be stopped for hatching, and the eggs should be re-hatched when the egg production rate returns to normal.

2 Immunization In the endemic area, eggs are inoculated with attenuated vaccines at 90 100 days of age, and breeders are vaccinated at 120 140 days of age. Avian encephalomyelitis virus oil emulsion can also be used to inactivate the vaccine intramuscularly. Eggs cannot hatch for 6 weeks after inoculation.

In non-epidemic areas, all inactivated seedlings are inactivated intramuscularly with avian encephalomyelitis virus oil emulsion. In the area of high incidence of the disease, can be used in combination with weak seedlings and oil milk seedlings, after the first immunization at the age of 70 days, laying hens at the age of 110 130 and then exempt, breeders at the age of 100 110 days and then exempt.

In some sources, the following suggestions are also made:

The disease mainly occurs in chicks within 4 weeks of age, the younger the age of infection, the more severe the symptoms, the higher the mortality rate, with the increase of age, the resistance gradually increases.

1. Do a good job of introducing eggs, and do not introduce eggs from epidemic areas. Eggs from previously diseased hens have reduced egg production, and eggs during this period may contain the virus, so they should not be reserved for seed use.

2 Sick flocks should be isolated, culled, and buried deeply, because horizontal transmission can occur between infected chickens and susceptible chickens or adult chickens.

3 Infected chickens and vaccinated chickens can develop strong resistance, and the resistance is passed on to the offspring through the yolk, so that the hatched chicks are resistant within 4-6 weeks.

1) live vaccine immunization The live vaccine currently used is still pathogenic to chicks, so it is advisable to give more than 10 weeks old reserve breeders to drink, nose drops or eye eyes for the whole group, or only to 5 chickens orally, through detoxification and cohabitation infection.

2) inactivated vaccine is best to use inactivated vaccines for chickens that are laying eggs, because live vaccines may affect egg production, it has been reported that 2 weeks after inoculating inactivated vaccines to 3-month-old chickens, the titer of the antibody of agar diffusion test is mostly above 1:4 for 9 months.

The two vaccines can be used alone or in combination. Attenuated vaccine inoculated at 8-10 weeks of age and prenatal vaccination; Inactivated vaccines are injected intramuscularly one month before the start of production; It is also possible to inoculate attenuated vaccines at 10-12 weeks of age, and then inoculate inactivated vaccines one month before the start of production.

Children with encephalomyelitis are mostly disseminated encephalomyelitis, which is a demyelinating disease. After the onset of the disease, it mainly damages the white matter of the brain and spinal cord, resulting in severe damage to the autonomic nerve. Magnetic resonance shocks are not obvious to the diagnosis of this disease, and the symptoms are sometimes somewhat similar to those of Guillain-Barre, if the delay will cause permanent innervation area dysfunction, and the re-damage of the delayed nerve will lead to further aggravation of the disease, and in severe cases, delayed ischemic cerebral atrophy or more serious conditions.

**Chinese and Western compound dialectical treatment, enhance the body's immune regulation function, and improve the body's ability to resist diseases. Nutrition of the nerves, the combination of traditional Chinese and Western medicine to expand the microcirculation so that the damaged residual nerves can get sufficient blood supply, to prevent the disease from further developing. At the same time, it excites the nerves, activates the nerve cells that are paralyzed and shocked, so that the body produces viral antibodies and no longer achieves the regeneration and repair of damaged nerves to obtain the best recovery of movement, stool and so on.

If you help send magnetic resonance** for your guidance.

Seriously, don't light isolation and feeding, feed antibiotics, antibiotics, antibacterial infections, vitamin E, vitamin B1 and gamma oryzanol, if you want me to say, if the number is small, just throw it away.

This disease is a problem with breeders, or is it acquired.

Encephalomyelitis has an incubation period of 53 131 days. It occurs mainly in lambs at 2 to 6 months of age. There is a clear seasonality, mostly occurring in March and August every year.

At the beginning, the sick sheep was depressed and lame, and then the four strands of rigidity or ataxia. Paralysis of one or more limbs, lying on the side, scribbling of limbs, nystagmus, panic, and reflexion of the angular arch in some cases. The head and neck are tilted or moved in circles.

Sometimes facial nerve paralysis, difficulty swallowing, or blindness. Proprioceptive examination is markedly absent, but motor neuron reflexes are not necessarily absent. Some knee or contraction reflexes are absent, but most are normal, and sometimes the muscles of the affected limb are markedly atrophied.

The prevention and treatment of this disease is mainly based on strengthening the management of breeding and adopting comprehensive health and epidemic prevention measures. Strengthen quarantine, prohibit the introduction of breeding sheep from the epidemic area, before the introduction of breeding sheep, should be seriological examination, after being transported back to quarantine for one year, during which two serological examinations (interval of half a year), are negative before the group can be mixed. This disease does not have much significance.

If necessary**, anti-inflammatory drugs (eg, aspirin, phenylbutazone, etc.) can be used to relieve local inflammation in the early stages of the disease, and broad-spectrum antibiotics can be given to prevent bacterial secondary infection.

Excuse me: What are the symptoms and methods of sheep inflammation?