Can neonatal cerebral hypoxia heal on its own, and how to care for it?

Neonatal cerebral hypoxia is mainly caused by the blow of hypoxia during or after childbirth. The degree of cerebral hypoxia depends on the degree of hypoxia, as follows: 1. General, such as keeping quiet, inhaling oxygen, keeping warm, keeping the respiratory tract open, correcting acidosis, hypoglycemia, hypocalcium, etc.

During the ** process, it is necessary to pay attention to observe the child's body temperature, respiration, consciousness, pupil size, fontanelle condition, and whether there are convulsive manifestations. If necessary, cardiopulmonary, blood pressure, intracranial pressure, and electroencephalogram (EEG) are monitored. If the child has a more severe convulsion, it is necessary to actively prevent cerebral edema and anticonvulsant**; 2. Late stage**:

Including hyperbaric oxygen**, hyperbaric oxygen chamber** can achieve good results in children with hypoxic-ischemic brain injury, because it can increase the blood oxygen content and partial pressure of blood oxygen in children, accelerate the clearance of lesions, and achieve the purpose of protecting the structure of the nervous system. Oxygen therapy can also provide sufficient oxygen supply to brain cells, accelerate the recovery of brain function, and drugs that promote brain cell metabolism can also be used.

There is no way, but you can slowly cultivate it in the later stage, and you must pay attention to the child's physical health in the later stage, and you must add more nutrients to the child, especially some nutrients for brain development.

It can't heal itself, you usually have to take some drugs, and you also need to inhale oxygen, you can't do strenuous exercise, you can put it in an incubator.

Cerebral hypoxia cannot be cured, because neonatal cerebral hypoxia is likely to lead to imbecile children, and they must be breastfed at ordinary times.

Abstract Dear, we have found out whether neonatal ischemic-hypoxic encephalopathy can be cured depends on the duration and severity of hypoxia in the child.

Neonatal ischemic-hypoxic encephalopathy is a brain lesion caused by hypoxia in perinatal neonates, and the common causes are intrauterine distress of the fetus caused by various reasons, such as umbilical cord around the neck, abnormal amniotic fluid, etc., and also common in the process of delivery and postnatal asphyxia and hypoxia, and a few can be seen in brain damage caused by other causes.

It usually occurs in full-term infants, but it can also occur in premature infants. The fetal heart rate may increase or slow during delivery, or the second stage of labor may be prolonged, the amniotic fluid may be contaminated with meconium, there is a history of asphyxia at birth, and there are still changes in consciousness, muscle tone, respiratory rhythm, etc., and even convulsions after resuscitation. Its clinical manifestations mainly depend on the duration and severity of hypoxia, and are mainly manifested as impaired consciousness, convulsions, intracranial hypertension, changes in muscle tone, changes in primitive reflexes, etc., and in severe cases, central respiratory failure.

Strengthening perinatal care, preventing perinatal asphyxia, and early active and correct resuscitation are the keys to preventing neonatal ischemic-hypoxic encephalopathy. Primarily supportive, symptomatic, post-neonatal and early intervention.

Can neonatal hypoxic-ischemic encephalopathy be a**?

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Dear, we have found out whether neonatal ischemic-hypoxic encephalopathy can be cured depends on the duration and severity of hypoxia in the child. Neonatal ischemic-hypoxic encephalopathy is a brain lesion caused by hypoxia in perinatal neonates, and the common causes are intrauterine distress of the fetus caused by various reasons, such as umbilical cord around the neck, abnormal amniotic fluid, etc., and also common in the process of delivery and postnatal asphyxia and hypoxia, and a few can be seen in brain damage caused by other causes. It usually occurs in full-term infants, but it can also occur in premature infants.

The fetal heart rate may increase or slow during delivery, or the second stage of labor may be prolonged, the amniotic fluid may be contaminated with meconium, there is a history of asphyxia at birth, and there are still changes in consciousness, muscle tone, respiratory rhythm, etc., and even convulsions after resuscitation. Its clinical manifestations mainly depend on the duration and severity of hypoxia, and are mainly manifested as impaired consciousness, convulsions, intracranial hypertension, changes in muscle tone, changes in primitive reflexes, etc., and in severe cases, central respiratory failure. Strengthening perinatal care, preventing perinatal asphyxia, and early active and correct resuscitation are the keys to preventing neonatal ischemic-hypoxic encephalopathy.

Primarily supportive, symptomatic, post-neonatal and early intervention.

We were born and lived in an incubator after the rescue, lived for 18 days, and have been in **, in the fight against mouse nerve growth factor, now we have been playing 5 courses, we have also done two courses of hyperbaric oxygen, from 3 months to do **, we have been **, pro, there will be miracles.

Severe ischemic-hypoxic encephalopathy in newborns is generally not good**, and very few children return to normal. In this case, it is necessary to carry out ** training as early as possible, do not delay the child's condition, pay attention to the appropriate rest of the child, pay attention to more ventilation in the child's room, and ensure a certain humidity and temperature.

Of course, it can be cured, as long as you actively cooperate with the doctor, you will be able to do it as soon as possible.