My child has chorea, what should I do?

Updated on healthy 2024-06-23
5 answers
  1. Anonymous users2024-02-12

    Chorea refers to Huntington's disease, which includes medications, training, etc., and is an inherited neurodegenerative disease characterized by chorea-like involuntary movements, mental disorders, and dementia, and is inherited in an autosomal dominant manner. 1. Drug**Dyskinesia is not serious, usually no drug is needed**, if there is a fall caused by a large range of movement, reserpine dopamine depleting agent can be used to control abnormal movement. In the case of muscle rigidity, spasticity, and abnormal muscle tone in the later stages, benzodiazepines such as clonazepam or antiparkinsonian drugs such as amantadine, levodopa, and carbidopa are commonly used**.

    If cognitive impairment occurs, neuroprotective drugs such as coenzyme Q10 and creatine may be considered. For patients with symptoms such as anxiety and depression, drugs such as escitalopram and sertraline can be used as prescribed**. Patients with severe and persistent hallucinations and delusions may be treated with low-dose second-generation antipsychotics, such as olanzapine and quetiapine.

    2. Training for Huntington's disease patients should start with balance and stability training from the early stage of the disease. Balance training activities can include exercises such as standing on one foot, walking in a straight line, and simple yoga and tai chi. At the same time, aerobic exercises such as brisk walking, jogging, and swimming can also be carried out outdoors or on a treadmill to strengthen cardiovascular function.

    It is important to note that all exercise activities need to be supervised at the beginning of the period to prevent the patient from falling. If your exercise program has been interrupted, you need to start at a lower intensity and gradually increase the intensity of your workout.

  2. Anonymous users2024-02-11

    Chorea may be genetic, and there are many symptoms, so can chorea be cured?

    Chorea is generally not curable, because chorea is a disease caused by congenital chromosomal abnormalities, which cannot be completely treated by drugs or surgery, and patients can choose to treat the symptoms. When a patient has some psychiatric symptoms, it needs to be carried out under the guidance of a doctor through the use of antipsychotic drugs**, and when the patient has dementia symptoms, it needs to be carried out by using drugs that nourish the brain nerves**.

    Traditional Chinese medicine pays attention to syndrome differentiation and treatment, if chorea belongs to the wind meridians, it is necessary to follow the principle of dispelling wind and channeling, nourishing blood and camping, and choose the best Chinese medicine for conditioning. If chorea belongs to the type of liver and kidney yin deficiency, it is necessary to nourish the liver and kidney, nourish the blood and quench the wind. Only in this way can the symptomatic selection of Chinese patent medicine achieve the best effect and alleviate the symptoms.

  3. Anonymous users2024-02-10

    Chorea is generally a neurological disorder. Most of them affect the central nervous system. Thinking should be affected. However, many diseases have similar symptoms. It is recommended that you go to the Department of Neurosurgery of Zhongshan Hospital Affiliated to Dalian University (Dalian Railway Hospital).

    In 1999, Zhongshan Hospital Affiliated to Dalian University (Dalian Railway Hospital) officially signed an agreement with Dalian Medical University to become the Fourth Affiliated Hospital of Dalian Medical University. At the same time, the hospital has established "sister hospital" relations with many hospitals at home and abroad, such as Osaka Neurosurgery Hospital in Japan, Suginda Eye Hospital in Japan, and Tongji Hospital affiliated to Tongji University in Shanghai. Persist in sending scholars to visit each other and carry out academic exchanges.

    Professor Tatsu Sugita Tatsu, President of Sugita Eye Hospital in Japan, regularly comes to our hospital for academic exchanges and surgeries. Currently, Professor Tatsu Sugita has been appointed as the Honorary Dean of our school. Professor Yoji Tatsumoto, President of the Japan Neurosurgery Hospital, insists on coming to our hospital regularly for consultations and brain surgery.

    Many physicians in the hospital have studied at home and abroad. Dean Professor Zhao Dewei has carried out academic exchanges and lectures in the United States, South Korea, Japan and other countries. Gao Baoshan, director of the Department of Neurosurgery, has studied in Japan for many years, and many doctors in the department have studied neurosurgery in Japan.

    Through the inter-hospital exchanges, the improvement of the medical technology level of the hospital has been greatly promoted.

  4. Anonymous users2024-02-09

    Huntington's disease is a dominantly inherited disorder that runs in families. Nerve cells in the brain continue to degenerate, and the body cells mistakenly produce a harmful substance called "huntingtin protein" due to a genetic mutation or an overexpansion of the CAG trinucleotide repeat in the DNA matrix of the fourth chromosome. These abnormal proteins accumulate in clumps and damage some brain cells, especially those involved in muscle control, leading to gradual degeneration of the nervous system, diffusion of nerve impulses, uncontrolled seizures, and the development of dementia and even death.

    Huntington's disease is a dominant genetic disorder with no specific effect**, and medication is palliative.

  5. Anonymous users2024-02-08

    In the acute phase of chorea, bed rest is required, and patients with psychiatric symptoms should be treated symptomatically. In this disease, hormones should be preferred, and oral prednisone should be preferred. Performed for rheumatism.

    folded hereditary progressive chorea; An autosomal dominant disorder with a predominantly occurring location in the Huntington's gene on chromosome 4, which is mainly caused by the decline of cholinergic and -aminobutyric acid neurons in the striatum, resulting in relatively hyperfunctional dopaminergic neurons in the substantia nigra. The main pathological manifestations are obvious loss of small intermediate cells in the caudate nucleus and putamen nucleus, accompanied by cerebral cortex atrophy and ventricular enlargement. The common age of onset is 30-45 years, but it is also occasionally seen in children.

    The first symptom is insidious involuntary movements, most pronounced on the face and upper extremities, often rapid and"Runiability"Dance-like moves. Progressive dementia usually occurs within years of the onset of involuntary movements, and most patients become sluggish, apathetic, or excited, and hallucinate. The disease is progressive and eventually leads to death.

    The disease usually lasts 10 to 15 years, and survival of 20 to 30 years is not uncommon. Haloperidol or chlorpromazine can reduce involuntary movements, but they do not affect the progression of dementia and disease.

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