What are the treatments for nephrotic syndrome?

Problem analysis: The typical symptoms of nephrotic syndrome are massive ovuria, hypertension, hyperazemia, high edema and hypoproteinemia.

Opinions and suggestions: To diagnose nephrotic syndrome, urine and blood tests should be carried out, and it is clear that a renal puncture should be done, and the pathological type should be clarified.

For mild nephrotic syndromes such as IgA nephropathy and grade, patients can choose ACE inhibitors or ARBs to reduce proteinuria while protecting the kidneys**. There are four regimens for more severe nephrotic syndrome: 1. Hormone**

Patients can choose oral hormones**, such as prednisone drugs, and patients should take one milligram per kilogram of body weight and take it in sufficient amount after meals; 2. Cytotoxic drugs: patients can choose cyclophosphamide oral or static point, and the impact ** is often static point; 3. Anticoagulation**: patients with nephrotic syndrome are prone to thrombosis in the renal microcirculation, and anticoagulation is an important means to control the development of thrombosis; 4. Traditional Chinese Medicine**:

Scientific and reasonable collocation** helps patients as soon as possible**.

Nephrotic syndrome**? Nephrotic syndrome is a disease caused by a variety of ** diseases with clinical features such as massive proteinuria, hypoproteinemia, edema, and hyperlipidemia. So how does nephrotic syndrome work?

First, the degree of lack of training.

Recovery of a patient's condition depends on the specifics and early stage of the patient's condition. Nephrotic syndrome, uremia will affect life safety if it is not **. Therefore, patients need to detect nephrotic syndrome at an early stage and target it**.

Second, avoid**.

To prevent nephrotic syndrome, attention should be paid to drugs, close coordination, early diagnosis and renal biopsy, to determine the nature of the disease at the level of cellular structure, and then to develop an individualized plan.

3. What is the problem of nephrotic syndrome? Choice of treatment method.

1.Glucocorticoids: inhibit the secretion of inflammatory response, immune response, aldosterone and antidiuretic hormone, affect the permeability of the glomerular basement membrane to diuresis, and eliminate urine protein due to the influence of comprehensive function.

2.Traditional Chinese medicine can reduce the use of hormones and cytotoxic drugs. Traditional Chinese medicine immunosuppressive drugs, drug selection: Tripterygium wilfordii polyglycoside Kunming Begonia, Kunming Mountain Begonia, Zhengqingfeng Tingning, increase liver albumin synthesis: Astragalus.

3. Diuresis and swelling: take thiazide potassium-sparing diuretics, and the effect is good. Poor effect, osmotic diuretic, albumin, diuretics.

4.The living environment of the patient should be kept clean in the indoor air and the place where the patient is not crowded, keep the destruction hole clean, prevent damage, prevent infection, and diagnose and treat infection in time. For digestion, eat lightly. Pay attention to physical and mental work, strengthen immunity, and exercise.

5.Patients should be on bed rest, advocate a diet with normal amounts of high-calorie and high-quality protein, and low-salt foods for edema; Eat foods with less animal oil and high cholesterol, such as egg yolks.

There are many factors that affect infection through primary nephrotic syndrome, the most common is infection, because in general, patients with nephrotic syndrome will have decreased immunity and decreased defense ability, such as bacteria, bacteria or fungi and other factors, which can easily lead to recurrent infection, the formation of immune complex deposition, increase the kidney damage of kidney cells, and lead to further fibrosis and sclerosis of the fibrous glomeruli. The hypercoagulable state of blood in patients with nephrotic syndrome is the result of damage to the renal parenchyma and is the cause of further promotion of nephropathy and refractory nephrotic syndrome. Nephrotic syndrome leads to further hypoxia-ischemic hypercoagulability of renal blood, which predisposes to thrombosis.