What are the specific characteristics of motor neurons?

Motor neuron usually has muscle weakness and atrophy of the hand as the first symptom, which generally starts from one side and then spreads to the other side, and with the development of the course of the disease, there is a mixed symptom of upper and lower motor neuron damage, which is called amyotrophic lateral sclerosis. The common symptoms of motor neuron are generally more severe damage to the lower motor neurons of the upper limbs, but the muscle tone can be increased, the tendon reflexes can be active, and there are pathological reflexes.

1.Onset of symptoms: In the early stages of the disease, you may not be able to hold chopsticks in your hands, or you may fall when you walk for no reason; It begins with a hoarse voice with no noticeable symptoms. Neuromuscular physicians should do the necessary tests such as electromyography, nerve conduction velocity, and magnetic resonance imaging to confirm the diagnosis.

2.Difficult period at work: Obvious limb weakness or even atrophy, life can still take care of themselves, but obstacles have appeared in the workplace.

At this time, it is necessary to take moderate rest to avoid aggravation of the disease. Necessary rehabilitation is provided by a rehabilitation physician and a social worker assists in psychological adjustment and social resources.

3.Difficult life: In the middle stage of the disease, the hands or feet, or the hands and feet have serious impairment at the same time, and they are unable to take care of themselves, and may not be able to walk, dress, hold chopsticks, and slurred speech.

4.Dysphagia stage: In the middle and late stages, the limbs are almost completely weak, the speech is severely impaired, it is easy to choke when eating, and some need to be fed, otherwise it is easy to lead to aspiration pneumonia.

5.Dyspnea: Respiratory muscles have been affected, breathing is difficult, patients may be bedridden, need to use a respirator, some will be admitted to a respiratory center** or choose home care, and some will be served by a hospice team.

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Motor neuron disease, generally with a family history, is mainly characterized by the above, and lower motor neuron involvement is the main manifestation, including muscle weakness, muscle atrophy, and increased muscle tone, hyperreflexia, positive pathological signs, generally no paresthesias, and urinary and bowel disorders.

Among them, muscle weakness, muscle atrophy, lower motor nervous system involvement, and hyperreflexia are the main manifestations of upper motor nervous system involvement.

Different patients will present with a variety of presenting symptoms, and most patients will have asymmetrical local, limb, and weak diseases.

For example, walking stiffness, easy to fall, finger movement is not flexible, can also be discharged, a small number of patients with normal respiratory system, the disease with the progression of the disease gradually muscle atrophy, extended to the whole body, in addition to eye activities, the whole body of the motor system, may be involved respiratory muscles will have dyspnea, respiratory failure, most patients may eventually die of respiratory failure.

Amyotrophic lateral sclerosis.

The most common first symptom is clumsiness and weakness in one or both fingers, followed by atrophy of the small finger muscles that progresses to the forearm, upper arm, and shoulder muscles. As the disease progresses, muscle weakness and atrophy extend to the trunk and neck, and eventually to the facial and laryngeal muscles. In a few cases, muscle atrophy and weakness begin in the lower extremities or trunk.

Fasciculations are often evident in the affected area.

Progressive amyotrophy.

The most common first symptom is atrophy and weakness of the small muscles in one or both hands, which gradually affects the muscles of the forearm, upper arm, and shoulder. Affected muscles are markedly atrophied, muscle tone is reduced, and muscle tremors may be seen.

Progressive bulbar palsy.

The main manifestations are progressive slurred articulation, dysphagia, choking on water, and chewing weakness. The tongue muscles are obviously atrophied, and there are fasciculations, and the lip muscles and throat muscles are atrophied. This type of disease progresses rapidly, and most of them die within 1 to 2 years due to respiratory muscle paralysis or lung infection.

Primary lateral sclerosis.

The most common presenting symptom is symmetrical stiffness and fatigue in both lower extremities, with a scissor gait in walking. Progresses slowly and gradually affects both upper extremities. There is usually no muscle atrophy and fasciculations, and no sensory deficits. This type progresses slowly and can survive for a long time.

In general, in adult animals, any external striated muscle fiber is innervated by only one oral motor neuron, so the motor unit is a functional unity and a quantum unit of motion. All myofibers innervated by a motor neuron exhibit the same histochemical properties and are thought to exhibit the same mechanical properties.

Motor neurons present clinically with varying combinations of muscle weakness, muscle atrophy, and bulbar palsy, such as dysphagia and slurred speech.

The early stages of MND begin with persistent weakness or spasms in one arm or leg and cause difficulty moving that part of the limb.

The first symptoms are clumsiness and weakness of one or both fingers, finger stiffness, followed by atrophy of the small muscles of the hand;

As the disease progresses, muscle atrophy extends to the forearms and lower limbs, and finally affects the face and throat, with significant fasciculations in the affected area;

In the advanced stage of the disease, weakness of tongue protrusion, difficulty swallowing, weakness in chewing, and slurred speech may occur;

In a few cases, muscle weakness and muscle atrophy may begin in the lower extremities;

Generally, there are no objective sensory disorders, but there are often subjective sensory disorders, such as numbness.

Progressive amyotrophy.

The first symptom is atrophy and stiffness of small muscles in one or both hands;

progressive involvement of the forearm, upper arm, and shoulder girdle muscles;

Affected muscle atrophy.

Noticeable, muscle tone.

decreased, fasciculations, tendon reflexes, and pathological reflexes may be seen;

Sensory system and sphincter dysfunction are generally absent.

Progressive bulbar palsy.

slurred pronunciation, hoarseness, dysphagia, choking on water, weakness in chewing, and loss of gag reflex;

Sensory system and sphincter dysfunction are generally absent.

Primary lateral sclerosis.

The first symptoms are symmetrical stiffness and fatigue in both lower extremities, and walking with a scissor gait.

Progresses slowly and gradually affects the upper extremities.

Muscle tone in the extremities is increased, and muscle atrophy and fasciculations are generally absent.

The sensory system and sphincter are not affected.

There are many different types of MND and their manifestations vary.

Clause. 1. Amyotrophic lateral sclerosis is manifested as insidious onset, progressive course, onset after the age of 40, men are a little more than women, and the average survival time is three to five years. The small motor movements of the hand are inflexible, the strength is weakened, the hand muscles atrophy and gradually affect the proximal end of the upper extremity, the tendon reflexes of the upper limbs are weakened or disappear, the lower limbs are spasmodic paralysis, there are occasional subjective sensory abnormalities, there is no objective sensory impairment, and the sphincter is involved.

Clause. 2. Progressive bulbar palsy, which begins after middle age, generally has a poor prognosis, and most of them die one to three years after the onset of the disease. There are unclear arthria, choking on drinking water, dysphagia, atrophy of tongue muscles, loss of gag reflex, and coexistence of true and false bulbar paralysis.

Clause. 3. Progressive musculospinal atrophy occurs in more men around the age of 30, and the first symptoms mostly start from the hands, and then the paralysis sensation and sphincter ability are normal, and gradually affect the entire upper limbs.

Clause. Fourth, the primary bridge grandson lateral sclerosis sclerosis.

Typical features, muscle weakness, muscle atrophy, and fasciculations are manifestations of lower motor nervous system involvement; Hypertonia, hyperreflexia, and positive pathological signs are the main manifestations of upper motor nervous system involvement. The presenting symptom can be a variety of manifestations depending on the patient. Most patients have asymmetrical local limb weakness, such as stiff walking, procrastination, easy falling, and inflexible finger movement.

Bulbar symptoms such as dysphagia and dysarthria. A small number of patients present with respiratory symptoms.

Muscle atrophy, muscle weakness, inability to **,

The main characteristics of motor neurons include:

1.Significant excitability: Motor neurons have such a high degree of excitability that can generate strong action potentials.

2.Fast conduction velocity: The nerve fibers of motor neurons have a fast conduction velocity and can quickly transmit nerve impulses to surrounding muscles.

3.High specificity: Each motor neuron can only control specific muscle fibers, therefore, motor neurons have a high degree of specificity for the control of muscle movement.

4.Intrinsic excitability: Motor neurons have a certain intrinsic excitability and can self-generate action potentials, which helps maintain muscle tone and posture. Closed Chunpei.

5.Vulnerability: The nerve fibers of motor neurons are susceptible to injury, and if they occur, they can lead to muscle weakness or paralysis.

Hello, patients with motor neuron disease mainly manifest as muscle atrophy, muscle weakness, fasciculations, increased muscle tone, tendon reflexia, paresthesias and urinary disorders, which are mainly manifestations of lower motor neuron involvement.

Symptoms of muscular dystrophy: infantile patients walk with a slow gait, easy to fall, delayed walking age, swaying pelvis from side to side when walking, heel can not touch the ground, Achilles tendon contracture, lordosis, back of the head, shoulder muscle atrophy, weakness, ** wrinkles, shoulder muscle atrophy, pterygoid shoulder deformity, pediatric patients have spine and limb joint deformities, later limb contractures, and complete loss of motor function. It is usually accompanied by lung infection, heart damage accompanied by symptoms such as palpitation, ulcers and bedsores, and a significant decrease in IQ in children.

It is still unclear that there are no specific medications.

Muscular dystrophy is a hereditary muscle degenerative disease, and there is no specific way, only symptomatic and supportive. It can increase the patient's nutrition, normal activities, physical ** and orthopedics, and can prevent and improve the deformation of the spine and the contracture of the joints. Because it is a lesion of the patient's muscles itself, strenuous exercise is not encouraged, and appropriate activities can be carried out to avoid long-term bed rest.

ATP, inosine, and vitamin E can be given orally or intravenously to provide energy support.

It's how fast you react to the exercise, if it's fast, it's your good, if it's slow, it's not good.

Motor neuron is also known as ALS, as the name of the disease suggests, patients will gradually appear as if they are "frozen", and the patient will gradually develop from limb weakness to muscle atrophy; The limbs gradually develop from the hands and feet, and eventually to the torso. Patients may experience dyspnea, dysphagia, and most eventually die of paralysis of the respiratory muscles. At present, Western medicine does not have a good method for motor neurons, but there is no way for motor neurons, and choosing the right professional method as soon as possible plays a vital role in motor neurons.

Motor neuron is an incurable disease, and the most important thing is to treat the symptoms**, so that it will slowly recover.