Can a cerebral infarction be cured, and what can be done to recover faster?

Can cerebral infarction be cured, this can be cured, but it takes time, because the formation of cerebral infarction is not overnight, it is a long-term accumulation process.

1. Cerebral thrombosis is a type of cerebral infarction.

2. The most fundamental and common cause of cerebral infarction is cerebral atherosclerosis, and the causes of cerebral atherosclerosis are very complex, and the common causes are: old age, diabetes, hyperlipidemia, hypertension, family history, high blood viscosity, etc. Predisposing factors:

Anger, over-agitation, over-exercise, exertion, cold, etc.

3. Therefore, cerebral infarction is possible, and with the increase of age, there are more and more chances. The key issue is ** and prevention.

4. The principle is: after the onset of the disease, it is necessary to race against time to send it to the hospital that can carry out thrombolysis, use aspirin or leech acid peptide, the blue packaging developed by Jingxuehuang, it is not recommended to use aspirin, because it is large, leech acid peptide is a strong anticoagulant substance, the effect on thrombolysis is very obvious or antithrombotic synthesis is carried out in hospitals with thrombolysis or antithrombolysis, the sooner the better, the sooner the better, this is the most critical point of cerebral infarction, if 95% of patients can be treated with satisfactory efficacy. If the disease is not timely or early failed to carry out a good antithrombotic synthesis, the sequelae are inevitable, and the drug can only improve after the future, and it is basically impossible to completely, if the course of the disease has passed for half a year, it will enter the sequelae period, and the effect will not be satisfactory.

5. Preventive measures: (1) Keep a good mood; (2) Appropriate amount of exercise; (3) Reasonable nutrition; (4) Have a regular schedule and rest; (5) Quit smoking and limit alcohol; (6) **Pre-existing diseases, such as diabetes, hyperlipidemia, hypertensive disease, etc.

After the occurrence of cerebral infarction, you should rush to the hospital, rush to the hospital within 63 6 hours of the onset of the disease, improve the relevant examinations, exclude thrombolysis contraindications, and give intravenous or arterial thrombolysis, so that the blocked blood vessels can be recanalized, and after the blood vessels are recanalized, the damaged nerve cells can be restored or the damage to the least extent, and the patient may return to normal.

For patients after more than 6 hours, antiplatelet **, anticoagulant**, and brain protection** can be given. If the patient has certain sequelae, a short-term or long-term plan can be formulated to carry out the first day, and some patients can also return to normal.

Cerebral infarction will leave many sequelae, such as monoplegia, hemiplegia, aphasia, etc., the effect of drugs on these cerebral infarctions is very limited, and through active and regular ****, most patients can achieve self-care, and some can return to work. If possible, it is best to go to a regular **hospital for the system**. **It is advisable to do it as early as possible.

3 to 6 months after the illness is the best time, half a year later due to the occurrence of cerebral infarction and joint contracture, the difficulty of ** is greater, but it will also help to a certain extent.

At present, acute cerebral infarction has become the most disabling and lethal disease in China. How to get better and faster in acute cerebral infarction mainly lies in the patient's admission to the hospital as soon as possible after the acute onset of cerebral infarction. In particular, intravenous thrombolysis can be given within 6 hours of onset**.

Intravenous drug thrombolysis** is mainly divided into two types: the first is Aitongli, which is imported from abroad, which is mainly used within the hour of onset. The second is the domestic drug urokinase, which is mainly useful within 6 hours of the onset of the disease.

Both of these drugs have a certain probability that thrombolysis is to block blood vessels and restore blood flow, so that the patient can regain certain limb function. However, none of them can be 100% successful, and if the patient's limb function does not improve after the application of the drug, endovascular intervention can be performed**. The first type of endovascular intervention**, mainly for endovascular stent thrombectomy**, is to use a stent to enter the body to reach the thrombus site, remove the thrombus from the body, and restore blood flow to the blocked blood vessels.

Second, if the patient has an acute cerebral infarction caused by the interruption of blood flow due to chronic vascular stenosis, endovascular stenting can be performed to restore the narrowed blood vessels, which can also lead to the recovery of the patient's neurological function, which can effectively save the patient's risk of disability and death.

It is necessary to go to the hospital as soon as possible for a detailed examination, according to the specific condition of the patient**.

Cerebral infarction often comes suddenly without warning, perhaps in sleep, at rest, or during exercise, which often leads to hemiplegia, unclear or even unable to speak, incontinence, and even abnormal mental behavior. Every patient with cerebral infarction and their families are looking forward to a faster recovery with no sequelae. Unfortunately, although the onset of cerebral infarction is sudden and rapid, the recovery process is very long.

Therefore, it is more appropriate to describe cerebral infarction as "illness comes like a mountain, and illness goes like a thread".

So, how to recover the fastest after cerebral infarction?

The recovery period is within one year of the onset of cerebral infarction, which is the most important period of cerebral infarction, and most patients have the fastest improvement and the best effect during this period. The most important thing for convalescent families is scientific medication, supplemented by functional training, dietary regulation and other comprehensive medications.

Only scientific medication can effectively treat the underlying lesions such as atherosclerotic plaque formation and high blood viscosity caused by cerebral infarction, prevent the continued formation of arteriosclerosis and thrombosis, and also create a good internal environment for brain tissue, which is also helpful for the recovery of the cerebral nervous system, and can improve the symptoms of cerebral infarction such as exercise and language.

Training is also very important for patients with cerebral infarction. Formal training can significantly reduce or alleviate the sequelae of paralysis. However, training is not the same as "exercising", and it needs to be carried out under the guidance of a doctor or a professional trainer.

**Training also needs to be gradual, not in a hurry, privately increase the intensity of **training, otherwise it will lead to joint muscle damage, foot sagging, foot inversion and other problems, severe cases can be fractured.

If patients do not receive good care and nutrition, it will seriously affect their quality of life and prognosis, and even endanger their lives. Therefore, it is also necessary to adjust the dietary structure for patients with cerebral infarction.

In general, the diet of patients with cerebral infarction should be different according to the severity of the disease, and the main intake is easy to digest, high in vitamins and low in fat. High-sugar, high-cholesterol, high-calorie foods should be avoided as much as possible, and attention should also be paid to controlling the addition of condiments, such as the content of salt is best controlled at 4-6g a day, and monosodium glutamate and the like should be avoided as much as possible. Many condiments contain salt, so be sure to add it in moderation.

Cerebral infarction is not "a single onset, lifelong immunity", but "a single onset, frequent recurrence", and the risk of recurrence after cerebral infarction is higher in the near future, and the risk of death is higher, the risk of disability is greater, and recovery is more difficult. Therefore, it is necessary to prevent stroke after cerebral infarction**!

**The best way for cerebral infarction is to clear the thrombus as soon as possible, dilate and dredge blood vessels, improve cerebral blood supply, reduce necrosis caused by lack of oxygen to brain cells as much as possible, restore corresponding functions, reduce sequelae, and improve the quality of daily life in the future. 1. Within 6 hours, try to be ultra-early**, at this time, the pathological changes of brain tissue are small, there is no obvious ischemic brain cell necrosis, the ischemic penumbra still exists, and it is in the time window of ischemia and reperfusion, and this period is the most critical. Thrombolysis, anticoagulation, fiber reduction, antiplatelet aggregation, blood dilution, and brain cell protection should be given based on the benefits and disadvantages**.

**The drug is: thrombolytic enzymes such as: pit viper antithrombose, urokinase, streptokinase, etc., the effect is excellent, but the drug has a fast and large reaction, which has the risk of internal bleeding, and the pros and cons of the drug should be mastered.

2. Early stage (6 72 hours): At this time, the ischemic penumbra has basically disappeared, thrombolysis** is meaningless, and fiber lowering and anticoagulation** can be tried;It is mainly anti-platelet agglutination and protection of brain cells**. Vasodilators plus blood-activating drugs such as:

Salvia japtiorrhiza needle, Chuanxiongzine needle, safflower needle, brevisanthin needle, etc., should not use vasodilators for those with increased intracranial pressure, and attention should be paid to reducing intracranial pressure. 3. Late acute phase (72 hours and 1 week): mainly antiplatelet agglutination and protection of brain cells, and drugs that protect brain cells and anti-cerebral hypoxia such as:

Cerebral protein hydrolysate injection, hemoactive injection and hyperbaric oxygen chamber oxygen inhalation. 4. Recovery period (2 years after 1 week): antiplatelet agglutination and protection of brain cells, mainly based on traditional Chinese medicine**

Qi deficiency and blood should be used with Brain Xuekang Oral Liquid, or Buyang Huanwu Tang plus or minus use, such as: Shengqi 20 40 grams, Angelica, Chuanxiong, Safflower, Peach Kernel, Dilong 12 grams each, Salvia miltiorrhizae 15 grams, leeches 12 grams, dry stool plus rhubarb, Citrus aurantium 10 15 grams each, Qi deficiency plus Codonopsis, Rebirth Qi, and with acupuncture, functional exercise, so that the combined use, the effect will be better.

1) Cerebral infarction should be carried out as soon as possible, as long as the patient is clear, the vital signs are stable, and the condition is no longer developing, it can be carried out after 48 hours, and the amount is from small to large, step by step. Most intracerebral hemorrhages** can begin 10 to 14 days after illness.

2) Mobilize the enthusiasm of patients.

**The essence is "learn, exercise, re-exercise, re-learn", which requires the patient to understand and actively engage. In the acute phase, ** exercise is mainly to inhibit abnormal primitive reflex activity and re-establish normal movement patterns, followed by training to strengthen muscles.

c) ** should go hand in hand with **.

Cerebral infarction is characterized by the coexistence of disorders and diseases, and an individualized approach is adopted, step by step. In addition to sports, attention should also be paid to speech, cognition, psychology, occupation and society. Some drugs, such as bromocriptine, have been shown to have a significant effect on the recovery of limb motor and speech function, and baclofen is effective in inhibiting spasticity, starting from small doses, and can be used as an option.

Clonidine, prazosin, phenytoin, diazepam, phenobarbital, and haloperidol adversely affect exercise in the acute phase and should be used sparingly or not at all.

4) Emphasize that ** is an ongoing process.

Closely observe stroke patients for depression and anxiety, which can seriously affect the progress and efficacy of the disease. Pay attention to the importance of community and family**.

Patients with mild cerebral infarction must pay attention to avoid eating greasy food and high-fat food in their daily diet, especially fried food, and can also appropriately apply some anticoagulant drugs to prevent the expansion of blood clots, such as aspirin, and at the same time pay attention to resting more and avoid fatigue. It is recommended to pay attention to the aura of cerebrovascular lesions in a timely manner, which is of great significance to the minor cerebral infarction.

Cerebral infarction is cerebral infarction, which is a disease caused by cerebral blood supply disorder and cerebral tissue ischemia and hypoxia, and the condition is more critical, so it needs to be treated as soon as possible to restore the cerebral blood supply as soon as possible, reduce the infarct area, and protect the non-infarcted brain tissue. Some patients with mild symptoms can obtain clinical **, which does not affect brain function, and if it has caused necrosis of brain tissue in important functional areas, it can generally not be cured, and brain function can only be improved as much as possible, and there may be various posterior limb sequelae.

1. Emergency treatment: If the patient has a sudden acute cerebral infarction and faints, call the emergency in time, try to avoid moving the patient too much, check the patient's mouth, take out dentures, foreign bodies, etc., gently lift the jaw, keep the airway unobstructed, and wait for the ambulance to arrive;

2. Thrombolysis**: cerebral infarction caused by arteriosclerosis, thrombosis and other reasons, in line with the indications, in the case of thrombolysis time window, intravenous infusion of thrombolytic drugs can be used for thrombolysis, relieve obstruction, and alleviate the symptoms of cerebral infarction.

3. Vascular intervention: stents can be placed in the blood vessels to improve vascular flow, or mechanical thrombectomy can be performed to improve blood flow and alleviate the symptoms of cerebral infarction;

4. Anticoagulation**: Patients in a hypercoagulable state can use antiplatelet aggregators or anticoagulant drugs to relieve blood or thrombus to continue to agglutinate and spread, and improve blood circulation;

5. Reduce intracranial pressure: make the state use mannitol, furosemide and other drugs to quickly reduce intracranial pressure, which is used to alleviate cerebral edema complicated with cerebral infarction;

6. Secondary prevention: Reasonable control of risk factors after cerebral infarction, such as hierarchical regulation of blood pressure, blood sugar and blood lipids, is an effective measure to prevent cerebral infarction.

Cerebral infarction is a common disease in middle-aged and elderly people, mostly caused by cerebral arteriosclerosis, stenosis and other reasons, the onset is acute, the condition is easy to recur, and it is easy to leave sequelae such as hemiplegia and poor activity, and the sequelae can be alleviated by traditional Chinese medicine acupuncture and other methods. Prompt medical attention in the event of cerebral infarction generally improves the prognosis, but long-term medication is required to prevent cerebral infarction**. If you have symptoms such as cerebral infarction, sudden speech impairment, numbness and weakness of one or one limb, you must seek medical attention as soon as possible.

Intravenous thrombolysis** can be used within 6 hours of the acute phase of cerebral infarction, especially within the hour of intravenous thrombolysis**.

Bridging** can also be used for large vessel occlusive lesions, i.e., intravascular thrombectomy and thrombectomy**. With aggressive and effective vascular recanalization**, approximately 1 in 3 patients can achieve rapid improvement or even complete remission.

If intravenous thrombolysis or arterial thrombectomy is missed, antiplatelet aggregation drugs, statins, and drugs that improve collateral circulation and protect brain mitochondria can also be used to promote neurological recovery.