Can severe ischemic hypoxic ischemic encephalopathy in newborns really be cured?

Severe ischemic-hypoxic encephalopathy in newborns is generally not good**, and very few children return to normal. In this case, it is necessary to carry out ** training as early as possible, do not delay the child's condition, pay attention to the appropriate rest of the child, pay attention to more ventilation in the child's room, and ensure a certain humidity and temperature.

Of course, it can be cured, as long as you actively cooperate with the doctor, you will be able to do it as soon as possible.

Abstract Dear, we have found out whether neonatal ischemic-hypoxic encephalopathy can be cured depends on the duration and severity of hypoxia in the child.

Neonatal ischemic-hypoxic encephalopathy is a brain lesion caused by hypoxia in perinatal neonates, and the common causes are intrauterine distress of the fetus caused by various reasons, such as umbilical cord around the neck, abnormal amniotic fluid, etc., and also common in the process of delivery and postnatal asphyxia and hypoxia, and a few can be seen in brain damage caused by other causes.

It usually occurs in full-term infants, but it can also occur in premature infants. The fetal heart rate may increase or slow during delivery, or the second stage of labor may be prolonged, the amniotic fluid may be contaminated with meconium, there is a history of asphyxia at birth, and there are still changes in consciousness, muscle tone, respiratory rhythm, etc., and even convulsions after resuscitation. Its clinical manifestations mainly depend on the duration and severity of hypoxia, and are mainly manifested as impaired consciousness, convulsions, intracranial hypertension, changes in muscle tone, changes in primitive reflexes, etc., and in severe cases, central respiratory failure.

Strengthening perinatal care, preventing perinatal asphyxia, and early active and correct resuscitation are the keys to preventing neonatal ischemic-hypoxic encephalopathy. Primarily supportive, symptomatic, post-neonatal and early intervention.

Can neonatal hypoxic-ischemic encephalopathy be a**?

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Dear, we have found out whether neonatal ischemic-hypoxic encephalopathy can be cured depends on the duration and severity of hypoxia in the child. Neonatal ischemic-hypoxic encephalopathy is a brain lesion caused by hypoxia in perinatal neonates, and the common causes are intrauterine distress of the fetus caused by various reasons, such as umbilical cord around the neck, abnormal amniotic fluid, etc., and also common in the process of delivery and postnatal asphyxia and hypoxia, and a few can be seen in brain damage caused by other causes. It usually occurs in full-term infants, but it can also occur in premature infants.

The fetal heart rate may increase or slow during delivery, or the second stage of labor may be prolonged, the amniotic fluid may be contaminated with meconium, there is a history of asphyxia at birth, and there are still changes in consciousness, muscle tone, respiratory rhythm, etc., and even convulsions after resuscitation. Its clinical manifestations mainly depend on the duration and severity of hypoxia, and are mainly manifested as impaired consciousness, convulsions, intracranial hypertension, changes in muscle tone, changes in primitive reflexes, etc., and in severe cases, central respiratory failure. Strengthening perinatal care, preventing perinatal asphyxia, and early active and correct resuscitation are the keys to preventing neonatal ischemic-hypoxic encephalopathy.

Primarily supportive, symptomatic, post-neonatal and early intervention.

We were born and lived in an incubator after the rescue, lived for 18 days, and have been in **, in the fight against mouse nerve growth factor, now we have been playing 5 courses, we have also done two courses of hyperbaric oxygen, from 3 months to do **, we have been **, pro, there will be miracles.

Infantile disease has infantile ischemic-hypoxic encephalopathy, so can infantile ischemic-hypoxic encephalopathy be cured?

Whether neonatal ischemic-hypoxic encephalopathy can be cured, and whether there are sequelae in the later stage, need to be determined according to the location of the baby's injury and the severity of the condition. In general, mild and moderate neonatal hypoxic-ischemic encephalopathy can be cured without sequelae. Severe neonatal ischemic-hypoxic encephalopathy is life-threatening in the early stage, and it is easy to have sequelae in the later stage, so it is necessary to follow up and re-examine in time, and if necessary, ****.

Once hypoxic-ischemic encephalopathy occurs, it is generally necessary to use drugs to nourish brain tissue to promote the improvement of brain function. In the future, you also need regular physical examinations to check the child's development, if there is a backwardness in development, especially in the big sports, you need to carry out training in time to promote the improvement of the overall development level.

Once perinatal asphyxia occurs, neonates are at high risk of hypoxic-ischemic encephalopathy. The health care provider needs to immediately perform a physical examination and** on the child. The purpose of neonatal hypoxic-ischemic encephalopathy** is to improve the metabolic function of damaged neurons as much as possible and maintain a stable internal environment; At the same time, it should be used to control convulsions, reduce cerebral edema, improve cerebral blood flow and brain cell metabolism.

1. General**:

1) Correction of hypotension: ensure adequate cerebral blood perfusion, commonly used dopamine 5-10 g kg per minute, intravenous infusion.

2) Correction of metabolic acidosis: sodium bicarbonate 2-3 meg kg 10% glucose diluted and slowly intravenous.

3) Correction of hypoxemia and hypercapnia: use an artificial respirator if necessary.

4) When the blood calcium is low, intravenous calcium gluconate can be used.

5) Provide sufficient glucose to meet the energy metabolism needs of brain tissue: 6-8mg kg per minute can be given.

6) Appropriately limit the amount of liquid: 50-60ml kg per day. The infusion rate is within 4ml kg h.

2. Control convulsions

Phenobarbital sodium is preferred, with the first dose of 15 to 20 mg kg, and if the convulsion is not resolved, it can be added 1 to 2 times at 5 mg kg each time, with an interval of 5 to 10 minutes, and the total load weight is 25 to 30 mg kg. The maintenance dose is 4-5 mg kg daily (once or in two divided doses) starting on day 2. It is best to monitor blood levels and discontinue the drug one week after the seizures have stopped.

If seizures are frequent, diazepam or chloral hydrate can be added.

3. Control increased intracranial pressure

Dexamethasone was used, furosemide 1 mg kg intravenously, and repeated after 4 to 6 hours. If the intracranial pressure is still high after 2-3 consecutive doses, switch to mannitol intravenously for 4-6 hours. Aim for a significant drop in intracranial pressure within 48 to 72 hours.

4. Central nervous system stimulants, etc

Cytochrome C, uraside triphosphate, coenzyme A, etc., can be intravenously instilled daily until the symptoms are significantly improved; Citicoline 100-125 mg per day can also be used, diluted and intravenous, and once a day intravenously starting on the second day after birth; 5ml of cerebrophyllin diluted with normal saline and intravenous drip can improve brain tissue metabolism.

**Must continue until symptoms completely disappear. Moderate HIE should be **10 to 14 days, and severe HIE should be **14 to 21 days or longer. **The sooner you start, the better, generally within 24 hours of birth**.

Try to avoid aggravating brain injury by various pathological factors after birth.